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Summary Uterine rupture is a well recognised and potentially fatal complication in the peripartum broodmare. This case report describes a 16 month history of infertility as the sole clinical sign in a Thoroughbred mare, subsequently diagnosed with a full thickness uterine tear. The clinical course, standing laparoscopically assisted repair and outcome are discussed.

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The hormonal milieu of mammals is in constant fluctuation throughout the life cycle, with progesterone (P4) and its metabolites serving as a primary constituent. In many species, P4 is believed to be highly anti-inflammatory, while a variety of synthetic progestins have been found to alter the immune response in comparison with that of progesterone. 1 Medroxyprogesterone acetate (MPA), levonorgestrel (LNG), noresthisterone acetate, and d-norgestrel have been shown to bind selectively to either the progesterone (PR) or glucocorticoid receptor (GR) on a variety of immune cell types, including macrophages, dendritic cells, natural killer cells, and eosinophils

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