Low temperature-induced fruit malformation in tomato was studied from plant hormonal view point. Treatment of TIBA (2, 3, 5-triiodobenzoic acid) and NAP (sodium N-1-naphthylphthalamic acid), auxin-transport inhibitors, increased locule numbers and induced severe malformation including catfaced and strawberry fruits with ovary splits, and oblate and fasciated fruits. Bendroquinone (2-benzimidoyl-3hydroxyl-1, 4-napthoquinone), which is considered to have the similar action mechanism to TIBA and NPA, also showed the same effects. Auxins, PCPA (p-chlorophenoxyacetic acid), 2, 4-D (2, 4-dichlorophenoxyacetic acid) and 2, 4, 5-T (2, 4, 5-trichlorophenoxyacetic acid) reduced oblate and triangular types of malformation by decreasing locule numbers. Gibberellin (GA3) induced ovary split but CCC [(2chloroethyl) trimethylammonium chloride] and SADH (succinic acid-2, 2-dimethylhydrazide), which are considered to inhibit gibberellin synthesis, could not reduce low temperature-induced fruit malformation.
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