The novel coronavirus SARS-CoV-2 (severe acute respiratory syndrome coronavirus-2) is the cause of the COVID-19 pandemic [
5
]. SARS-Cov-2 demonstrates partial resemblance to SARS-CoV and MERS-CoV in phylogenetic analysis, clinical manifestations, and pathological findings [
6
, 7]. Reports emerging from China have described ataxia as a neurological symptom of the SARS-CoV-2 infection [
5
]. Opsoclonus consists of back-to-back multidirectional conjugate saccades without an inter-saccadic interval [
8
]. Myoclonus is defined as a sudden, brief, “shock-like”, nonepileptic involuntary movement [
9
], which has been described as a symptom of SARS-CoV-2 infection [
10
]. Opsoclonus-Myoclonus-Ataxia syndrome (OMAS) associated COVID-19 infection has been reported recently [
11
12
].
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