Zn-air battery is considered as one of the most promising candidates for next-generation batteries for energy storage due to safety, high energy density, and low cost. There are many challenges in electrolytes for developing high-performance rechargeable Zn-air cells as well as electrocatalysts. An electrolyte is the crucial part of the rechargeable Zn-air batteries that determine their capacity, cycling stability, and lifetime. This paper reviews the most recent progress in designing and fabricating electrolytes in aqueous and flexible Zn-air batteries. The discussion on the surface reaction relationships was covered between air-catalyst-electrolyte and electrolyte-zinc reaction mechanism. We highlight the recent developments of three different electrolytes in zinc-air battery: aqueous electrolyte, room temperature ionic liquid, and quasi-solid flexible electrolyte. Furthermore, the general perspective is proposed for designing and fabricating electrolytes to improve the performance and prolong the lifetime of Zn-air batteries.

No abstract

No abstract

In present study, the feasibility of partial nitrification (PN) process achievement and its greenhouse gas emission were evaluated in a sequencing batch biofilm reactor (SBBR). After 90days' operation, the average effluent NH-N removal efficiency and nitrite accumulation rate of PN-SBBR were high of 98.2% and 87.6%, respectively. Both polysaccharide and protein contents were reduced in loosely bound extracellular polymeric substances (LB-EPS) and tightly bound EPS (TB-EPS) during the achievement of PN-biofilm. Excitation-emission matrix spectra implied that aromatic protein-like, tryptophan protein-like and humic acid-like substances were the main compositions of both kinds of EPS in seed sludge and PN-biofilm. According to typical cycle, the emission rate of CO had a much higher value than that of NO, and their total amounts per cycle were 67.7 and 16.5mg, respectively. Free ammonia (FA) played a significant role on the inhibition activity of nitrite-oxidizing bacteria and the occurrence of nitrite accumulation.

No abstract

SPDF and EIMW act as efficient anti-aggregation π-spacers due to the rigid ‘T’ configuration of the former and the large steric hindrance of the latter.

Hypoxia, the hallmark of malignant tumors, has been recognized as a major obstacle for photodynamic therapy (PDT). Precisely targeting cancer cells in intricate biological scenarios by a hypoxia-resistant photosensitizer (PS) is the cornerstone to conquer the inevitable tumor recurrence and metastasis. Herein, we describe an organic NIR-II PS (TPEQM-DMA) possessing potent type-I phototherapeutic efficacy to overcome the intrinsic pitfalls of PDT in combating hypoxic tumors. TPEQM-DMA exhibited prominent NIR-II emission (>1000 nm) with an aggregation-induced emission feature and efficiently produced superoxide anion and hydroxyl radical in the aggregate state under white light irradiation exclusively through a low-O2-dependent type-I photochemical process. The suitable cationic nature assisted TPEQM-DMA to accumulate in cancerous mitochondria. Meanwhile, the PDT of TPEQM-DMA impaired the cellular redox homeostasis, led to the mitochondrial dysfunction, and raised the level of lethal peroxidized lipids, which induced cellular apoptosis and ferroptosis. This synergistic cell death modality enabled TPEQM-DMA to suppress the growth of cancer cells, multicellular tumor spheroids, and tumors. To improve the pharmacological properties of TPEQM-DMA, TPEQM-DMA nanoparticles were prepared by encapsulation of polymer. In vivo experiments proved the appealing NIR-II fluorescence imaging-guided PDT effect of TPEQM-DMA nanoparticles for tumors.

The active free radical reaction in the gastric tissues can induce the carcinogenesis of non-MGC. The utmost low ability of antioxidation in the gastric tissues can induce the carcinogenesis of MGC. The metabolic change of the free radicals centralized mostly in the center of ulcerated lesions only, which suggested the ability of antioxidation was declined only in these lesions. However, the metabolism of free radicals varied significantly and the ability of antioxidation declined not only in the local focus area but also in the abroad gastric tissues with gastric carcinoma.