On the basis of predictions of the mood-behavior model (G. H. E. Gendolla, 2000) and motivational intensity theory (J. W. Brehm & E. A. Self, 1989), the authors conducted 2 studies that critically tested the common assumption that dysphoria is associated with a motivational deficit. Dysphoric and nondysphoric undergraduates performed a cognitive task that was either easy or difficult. Effort intensity (i.e., resource mobilization) was assessed as performance-related cardiovascular reactivity. In support of the authors' predictions and in contrast to the popular view of a general motivational deficit, both studies found a crossover interaction between dysphoria and task difficulty: In the difficult condition, nondysphoric participants indeed showed stronger systolic blood pressure reactivity than dysphoric participants. But in the easy condition, dysphoric participants showed stronger systolic reactivity than nondysphoric participants. The findings are discussed with respect to motivational deficits in depression and possible underlying mechanisms.

The White Bear Suppression Inventory (WBSI; Wegner & Zanakos, 1994) was originally designed to assess people's inclination toward thought suppression. In this article, we provide a detailed review of previous findings on the structure of this instrument and present a study that took a new statistical approach. It involved an exploratory factor analysis of the French WBSI using the weighted least squares mean and variance estimator as well as parametric item response theory analyses. Results clearly supported a 2-factor structure with a "suppression" and an "intrusion" dimension. Follow-up regression analyses revealed that intrusion significantly predicted anxiety and depression scores, whereas suppression did not.

Instigated by evidence for reduced responsiveness to reward in depression, the present two studies addressed the question if such anhedonic behavior would also become evident in reduced mobilization of mental effort in terms of cardiovascular reactivity. Undergraduates completed the Center for Epidemiologic Studies-Depression Scale (CES-D) and worked on mental tasks, expecting either no consequence, a performance-contingent reward, or a performance-contingent punishment. Study 1 revealed that participants with low CES-D scores showed high systolic blood pressure reactivity in the punishment condition, whereas participants with high CES-D scores showed low systolic reactivity. Study 2 corroborated this finding for reward: Nondysphoric participants expecting a reward showed higher reactivity of systolic blood pressure and pre-ejection period than participants in the neutral condition or than dysphoric participants. Together, the studies demonstrate that reward insensitivity in (subclinical) depression is also found in cardiovascular reactivity. Furthermore, dysphoric individuals do not respond to punishment either, suggesting a general insensitivity to hedonic consequences.

Two studies examined the influence of dysphoria on motivational intensity in a student sample. Participants worked on a memory task (Study 1) or a mental concentration task (Study 2) without fixed performance standard ("do your best"). Based on their scores on the Center for Epidemiologic Studies -Depression Scale (L. S. Radloff, 1977), dysphoric and nondysphoric students were compared with regard to their effort-related cardiovascular reactivity during task performance. As predicted on the basis of the mood-behavior-model (G. H. E. Gendolla, 2000) and motivational intensity theory (J. W. Brehm & E. A. Self, 1989), dysphoric participants showed stronger cardiovascular reactivity while working on the cognitive tasks than nondysphoric participants. In Study 1, nondysphoric participants performed better on the memory task than dysphoric participants. Theoretical implications are discussed.

Reward insensitivity in depression and dysphoria has been demonstrated by selfreport, behavioral, and neuroscience data. These findings show less anticipated and experienced pleasure to rewarding stimuli, no behavioral adaptation in anticipation of rewards, and altered functioning in reward-related brain areas. The present study expands previous research by using cardiovascular reactivity to three levels of reward as an indicator of effort mobilization. Undergraduates with low versus high depression scores worked on a cognitive task in anticipation of no, versus a small, versus a significant amount of money for successful task performance. Results of pre-ejection period and heart rate reactivity confirmed the expected linear increase as a function of reward value in nondysphoric participants and the expected blunted response across all reward levels in dysphoric participants. The present findings thus show that dysphoric individuals have a motivational deficit in terms of reduced effort-related cardiac reactivity when anticipating a monetary reward.