Summary Systemic acquired resistance (SAR), a natural disease response in plants, can be induced chemically. Salicylic acid (SA) acts as a key endogenous signaling molecule that mediates SAR in dicotyledonous plants. However, the role of SA in monocotyledonous plants has yet to be elucidated. In this study, the mode of action of the agrochemical protectant chemical probenazole was assessed by microarray‐based determination of gene expression. Cloning and characterization of the most highly activated probenazole‐responsive gene revealed that it encodes UDP‐glucose:SA glucosyltransferase (OsSGT1), which catalyzes the conversion of free SA into SA O‐β‐glucoside (SAG). We found that SAG accumulated in rice leaf tissue following treatment with probenazole or 2,6‐dichloroisonicotinic acid. A putative OsSGT1 gene from the rice cultivar Akitakomachi was cloned and the gene product expressed in Escherichia coli was characterized, and the results suggested that probenazole‐responsive OsSGT1 is involved in the production of SAG. Furthermore, RNAi‐mediated silencing of the OsSGT1 gene significantly reduced the probenazole‐dependent development of resistance against blast disease, further supporting the suggestion that OsSGT1 is a key mediator of development of chemically induced disease resistance. The OsSGT1 gene may contribute to the SA signaling mechanism by inducing up‐regulation of SAG in rice plants.

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The paper proposes the MIMO waveforms consisted of Doppler-offset orthogonal complementary codes (DO-OCC) for realizing high update rate MIMO system. The DO-OCC waveforms have low cross-correlation among multiplexed waves and low autocorrelation peak side-lobe level (PSL) even in a channel fluctuation condition. Computer simulation and experimental measurements have been done for verification. Consequently, our work proves the effectiveness of the proposed MIMO waveforms for high update rate MIMO radars systems.