The neutron longitudinal and transverse asymmetries A n 1 and A n 2 have been extracted from deep inelastic scattering of polarized electrons by a polarized 3 He target at incident energies of 19.42, 22.66 and 25.51 GeV. The measurement allows for the determination of the neutron spin structure functions g n 1 (x; Q 2 ) and g n 2 (x; Q 2 ) over the range 0:03 < x < 0:6 at an average Q 2 of 2 (GeV=c) 2 . The data are used for the evaluation of the Ellis-Ja e and Bjorken sum rules. The neutron spin structure function g n 1 (x; Q 2 ) is small and negative within the range of our measurement, yielding an integral R 0:6 0:03 g n 1 (x)dx = 0:028 0:006 (stat) 0:006 (syst). Assuming Regge behavior at low x, we extract n 1 =

Global polarization of Λ hyperons has been measured to be of the order of a few tenths of a percent in Au+Au collisions at √ s N N = 200 GeV, with no significant difference between Λ andΛ.These new results reveal the collision energy dependence of the global polarization together with the results previously observed at √ s N N = 7.7 -62.4 GeV and indicate noticeable vorticity of the medium created in non-central heavy-ion collisions at the highest RHIC collision energy. The signal is in rough quantitative agreement with the theoretical predictions from a hydrodynamic model and from the AMPT (A Multi-Phase Transport) model. The polarization is larger in more peripheral collisions, and depends weakly on the hyperon's transverse momentum and pseudorapidity η H within |η H | < 1. An indication of the polarization dependence on the event-by-event charge asymmetry 3 is observed at the 2σ level, suggesting a possible contribution to the polarization from the axial current induced by the initial magnetic field. PACS numbers: 25.75.-q, 25.75.Ld

The spin structure function of the neutron gr has been determined over the range 0.03 < x < 0.6 at an average Q2 of 2 (GeV/c)2 by measuring the asymmetry in deep inelastic scattering of polarized electrons from a polarized 3He target at energies between 19 and 26 GeV. The integral of the neutron spin structure function is fo. nd to be Ji g?(x) dx =-0.022 f 0.011. Earlier reported proton results 2. .- .-_together with the Bjorken sum rule predict &r g?(x) dx =-0.059 f 0.019.

Moments (Variance (σ 2 ), Skewness(S), Kurtosis(κ)) of multiplicity distributions of conserved quantities, such as net-baryon,net-charge and net-strangeness, are predicted to be sensitive to the correlation length of the system and connected to the thermodynamic susceptibilities computed in Lattice QCD and Hadron Resonance Gas (HRG) model. In this paper, we present several measurement artifacts that could lead to volume fluctuation and autocorrelation effects in the moment analysis of net-proton multiplicity distributions in heavy-ion collisions using the UrQMD model. We discuss methods to overcome these artifacts so that the extracted moments could be used to obtain physical conclusions. In addition we present methods to properly estimate the statistical errors in moment analysis.

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Recently Tevatron released their measurements on invariant mass spectrum of electron/positron, as well as the di-jet arising from WW+WZ production with one W leptonically decay. Though the statistics is not significant, there are two bumps around 240 GeV and 120-160 GeV respectively. We proposed that the two bumps correspond to the extra light gauge bosons Z ′ and W ′ , which couple with quarks with the deci-weak strength.In this brief report, we also simulated di-jet invariant mass distribution at the current running LHC.

A fundamental question regarding autophagosome formation is how the shape of the double-membrane autophagosomal vesicle is generated. Here we show that in mammalian cells assembly of an actin scaffold inside the isolation membrane (the autophagosomal precursor) is essential for autophagosomal membrane shaping. Actin filaments are depolymerized shortly after starvation and actin is assembled into a network within the isolation membrane. When formation of actin puncta is disrupted by an actin polymerization inhibitor or by knocking down the actin-capping protein CapZβ, isolation membranes and omegasomes collapse into mixed-membrane bundles. Formation of actin puncta is PtdIns(3)P dependent, and inhibition of PtdIns(3)P formation by treating cells with the PI(3)K inhibitor 3-MA, or by knocking down Beclin-1, abolishes the formation of actin puncta. Binding of CapZ to PtdIns(3)P, which is enriched in omegasomes, stimulates actin polymerization. Our findings illuminate the mechanism underlying autophagosomal membrane shaping and provide key insights into how autophagosomes are formed.