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Background: Synapse formation and function is modulated by intrinsic and extrinsic non-autonomous factors.Results: Astrocytes induce synapse formation through TGF-β1 pathway. TGF-β1 synaptogenic property is dependent on d-serine signaling.Conclusion: TGF-β induces excitatory glutamatergic synapses in vertebrates.Significance: This is a novel molecular mechanism that might impact synaptic function and shed light on new potential therapeutic targets for synaptic deficit diseases.

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Using purely syntactical arguments, it is shown that every nontrivial pseudovariety of monoids contained in DO whose corresponding variety of languages is closed under unambiguous product, for instance DA, is local in the sense of Tilson.