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In seismology, the rupture mechanisms of an earthquake, a glacier stick‐slip and a landslide are not directly observed, but inferred from surface measurements. In contrast, laboratory experiments can illuminate near field effects. The near field reflects the rupture mechanism but is highly attenuated in the case of real‐world surface data. We directly image the elastic wave‐field of a nucleating rupture non‐invasively in its near‐field with ultrasound speckle correlation. Our imaging yields the particle velocity of the full shear wave field at the source location and inside the 3D frictional body. We experimentally show that a strong bimaterial contrast, as encountered in environmental seismology, yields a unidirectional or linear force mechanism for pre‐rupture microslips and decelerating supershear ruptures. A weak contrast, characteristic for earthquakes, generates a double‐couple source mechanism for sub‐Rayleigh ruptures, sometimes preceded by slow deformation at the interface. This deformation is reproduced by the NF of a unidirectional force.

In shear wave elastography, rotational wave speeds are converted to elasticity measures using elastodynamic theory. The method has a wide range of applications and is the gold standard for non-invasive liver fibrosis detection. However, the observed shear wave dispersion of in vivo human liver shows a mismatch with purely elastic and visco-elastic wave propagation theory. In a laboratory phantom experiment we demonstrate that porosity and fluid viscosity need to be considered to properly convert shear wave speeds to elasticity in soft porous materials. We extend this conclusion to the clinical application of liver stiffness characterization by revisiting in vivo studies of liver elastography. To that end we compare Biot’s theory of poro-visco-elastic wave propagation to Voigt’s visco-elastic model. Our results suggest that accounting for dispersion due to fluid viscosity could improve shear wave imaging in the liver and other highly vascularized organs.

In seismology, the rupture mechanism of an earthquake, a glacier stick-slip and a landslide is not directly observed, but inferred from surface measurements. In contrast, laboratory experiments can illuminate near field effects, which reflect the rupture mechanism but are highly attenuated in the case of real-world surface data. We directly image the elastic wave-field of a nucleating rupture non-invasively in its near-field with ultrasound speckle correlation. Our imaging yields the particle velocity of the full shear wave field at the source location and inside the 3D frictional body. We experimentally show that a strong bimaterial contrast, as encountered in environmental seismology, yields a unidirectional or linear force mechanism for pre-rupture microslips and decelerating supershear ruptures. A weak contrast, characteristic for earthquakes, generates a double-couple source mechanism for sub-Rayleigh ruptures, sometimes preceded by slow deformation at the interface. This deformation is reproduced by the near field of a unidirectional force.

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In most elastography experiments, shear waves are generated using a single source on the surface with a shaker, or in the bulk with radiation pressure of ultrasound. However, emitting controlled shear waves from multiple sources is a good way to improve the signal to-noise-ratio for shear-wave elastography. The experiments are conducted using six shakers with independent driving electronics in gelatin-graphite to mimic the tissue. Based on time reversal, our approach shows the feasibility of controlling shear-wave field in space with multiple focal spots at chosen locations, and in time with a chosen delay between each focusing. Improved by 10 dB compared to the use of a single source, the signal-to-noise ratio demonstrates that time-reversal as an adaptive filter is a good method to deliver maximum energy vibrations toward deep regions. Furthermore, this adaptive approach allows controlled vibrations to be delivered through bone conduction: a shear-wave focal spot is experimentally observed in a soft brain tissue-mimicking phantom using the multiple sources array applied to a skull model.

We recently showed that the observed shear wave dispersion in a soft, porous, water-saturated tissue can be explained by Biot’s theory of poroelasticity. The theory explains the shear wave velocity increase with frequency due to a relative movement between the solid and the viscous fluid. We propose that fluid-solid interaction explains the observed shear wave dispersion in the liver, a naturally saturated organ. The liver is drawn through by a network of blood vessels and exposes a total porosity of about 14% [1]. Blood viscosity changes from patient to patient and depends on different factors such as hydration and fitness. We included the blood viscosity for a 14% porosity liver into the elasticity estimation from shear wave speed measurements for a given shear wave elastography dataset [2]. For 11 out of 50 patients, the fibrosis classification would change if blood viscosity is included. [1] C. Debbaut et al., “Perfusion characteristics of the human hepatic microcirculation based on three-dimensional reconstructions and computational fluid dynamic analysis,” J. Biomech. Eng. 134(1), 011003 (2012). [2] Jang et al., “Hemorheological alteration in patients clinically diagnosed with chronic liver diseases,” J. Korean Med. Sci. 31(12), 1943–1948 (2016).

<p>At the field scale and in most laboratory studies the rupture nucleation mechanism of an earthquake, landslide or glacier stick-slip cannot be directly imaged. Near-field and source effects are thus difficult to observe. We use correlation of highspeed ultrasound images to track shear wave propagation at the rupture nucleation source and in its near-field. The particle velocity and accumulated displacement of the shear wave field emitted by the rupture are observed in-situ on a very dense grid. The grid consists of the ultrasonic imaging plane inside the frictional body and resolution is defined by the ultrasonic wavelenth (0.3 mm). The rupture process is generated by controlling a driving slab through a motor and a granular layer of sand or gravel constitutes the stick-slip behavior. The frictional body is a homemade Poly-Vinyl-Alcohol hydrogel. Although its properties are differing from those of classically investigated rocks, it constitutes a linear elastic material and reproduces rupture processes that are known from the field and rock physics. Through the elastic wave field we observe microslips which precede supershear rupture propagation along the frictional interface. We experimentally show that the source mechanism of a breaking asperity depends on the material contrast of the adjacent halfspaces. Neither a double-couple nor a single-force mechanism perfectly reproduce the experimental data of a rupturing asperity, while micro-slips are well reproduced by a singular shear point force.</p>