These results suggest that the ACTN3 gene may play a significant role in determining muscle phenotypes. However, this gene is only one of many factors which could contribute to athletes' performance and muscle phenotypes.

ObjectivesThe aim was to examine the association between serum vitamin D concentration and isometric strength of various muscle groups, vertical jump performance, and repeated sprint ability in young ice hockey players. The secondary aim was to determine the association between vitamin D deficiency and indices of iron status.MethodsFifty male ice hockey players (17.2±0.9 years) participated in this cross-sectional study. Exercise performance was evaluated using isometric strength measures of upper and lower extremities, vertical jump performance and repeated sprint ability (RSA). Blood samples were collected for the determination of serum 25-hydroxyvitamin D (25(OH)D) and multiple indicies of iron status.ResultsThe mean serum 25(OH)D concentration was 30.4 ng·ml-1 and ranged from 12.5 to 91.4 ng·ml-1. Eleven participants (22%) had vitamin D deficiency and 20 athletes (40%) had vitamin D insufficiency. Serum 25(OH)D concentration was not positively correlated with isometric muscle strength, vertical jump performance, or RSA after adjusting for age, training experience, fat mass, fat free mass and height. Serum 25(OH)D concentration was not associated with indices of iron status.ConclusionVitamin D insufficiency is highly prevalent in ice hockey players, but 25(OH)D concentration but it is not associated with exercise performance or indices of iron status.

SummaryStudy aim: the aim of the study was to compare two methods for measuring punching and kicking force and the reaction time of athletes. Material and methods: both systems were designed to measure and to analyse the mechanical characteristics of punches and strikes delivered by upper and lower limbs to a punching bag. The main difference between both punching bags was the way in which the delivered force was measured. The first method used strain gauges while the second method used accelerometer technology. Both systems consisted of a punching bag with software, attached signal diodes, and either embedded accelerometers or strain gauges. The bags were of different sizes. Acceleration transducers and gyroscopes or strain gauges were placed inside the punching bags, which allowed for measuring dynamics while the bag was struck. The software calculated strike force, the point of force application and its direction, and reaction time. Both systems were tested. Results: the results of the accelerometer-based method show that the mean relative error of force calculation amounts to 3%. The measurement error of acceleration is less than 1%. The mean relative measurement error of the striking surface on the punching bag is 2%. However, the measurement error of force recorded with the strain gauge-based method is less than 1%. The results show that both systems are similar. Conclusions: the punching bag having an embedded accelerometer is equipped with more versatile software, which makes the system a good tool for practical application in combat sport training.

The identification of the vitamin D receptor in tissues related to testosterone and cortisol production, in conjunction with the observed correlations between vitamin D levels and these hormones in the general population, suggest vitamin D may influence testosterone and cortisol concentrations in athletes. A cross-sectional study design was used to evaluate the association between 25(OH)D and testosterone and cortisol concentrations in young male ice hockey players (n = 50). All athletes were recruited during October from the Sosnowiec area, Poland (50° N). Commercially available ELISA kits were used to determine total serum 25(OH)D, testosterone and cortisol concentrations. Serum 25(OH)D concentration was analyzed as both a continuous and dichotomous variable, binned at the criteria for deficiency (< 20 ng·ml-1), to investigate a threshold effect. Neither continuous (r = 0.18, p = 0.20) nor dichotomous (r = 0.16, p = 0.27) 25(OH)D concentration was significantly correlated with testosterone concentration. A small, inverse correlation (r = -0.30, p = 0.04) was detected between 25(OH)D and cortisol concentrations when analyzed as a dichotomous variable only. Serum 25(OH)D concentration was neither associated with testosterone (p = 0.09) nor cortisol concentrations (p = 0.11) after adjusting for age, fat free mass and fat mass in sequential linear regression. The inability of vitamin D status to independently predict testosterone and cortisol concentrations suggests that any performance-enhancing effects of vitamin D in athletes are unlikely to be mediated primarily through these hormones, at least amongst young male ice-hockey players.

Orysiak, J, Mazur-Różycka, J, Busko, K, Gajewski, J, Szczepanska, B, and Malczewska-Lenczowska, J. Individual and combined influence of ACE and ACTN3 genes on muscle phenotypes in polish athletes. J Strength Cond Res 32(10): 2776-2782, 2018-The aim of this study was to examine the association between angiotensin-converting enzyme (ACE) and α-actinin-3 (ACTN3) genes, independently or in combination, and muscle strength and power in male and female athletes. The study involved 398 young male (n = 266) and female (n = 132) athletes representing various sport disciplines (ice hockey, canoeing, swimming, and volleyball). All were Caucasians. The following measurements were taken: height of jump and mechanical power in countermovement jump (CMJ) and spike jump (SPJ), and muscle strength of 10 muscle groups (flexors and extensors of the elbow, shoulder, hip, knee, and trunk). The insertion-deletion (I/D) polymorphism of ACE and the R577X polymorphism of ACTN3 were typed using polymerase chain reaction (PCR) and polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP), respectively. The genotype distribution of the ACE and ACTN3 genes did not differ significantly between groups of athletes for either sex. There was no association between ACE and ACTN3 genotypes (alone or in combination) and sum of muscle strength, height of jump or mechanical power in both jump tests (CMJ and SPJ) for male and female athletes. These findings do not support an influential role of the ACE and ACTN3 genes in determining power/strength performance of elite athletes.