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Aims The aim of this study was to clarify the factors that predict the development of avascular necrosis (AVN) of the femoral head in children with a fracture of the femoral neck. Patients and Methods We retrospectively reviewed 239 children with a mean age of 10.0 years (sd 3.9) who underwent surgical treatment for a femoral neck fracture. Risk factors were recorded, including age, sex, laterality, mechanism of injury, initial displacement, the type of fracture, the time to reduction, and the method and quality of reduction. AVN of the femoral head was assessed on radiographs. Logistic regression analysis was used to evaluate the independent risk factors for AVN. Chi-squared tests and Student’s t-tests were used for subgroup analyses to determine the risk factors for AVN. Results We found that age (p = 0.006) and initial displacement (p = 0.001) were significant independent risk factors. Receiver operating characteristic (ROC) curve analysis indicated that 12 years of age was the cut-off for increasing the rate of AVN. Severe initial displacement (p = 0.021) and poor quality of reduction (p = 0.022) significantly increased the rate of AVN in patients aged 12 years or greater, while in those aged less than 12 years, the rate of AVN significantly increased only with initial displacement (p = 0.048). A poor reduction significantly increased the rate of AVN in patients treated by closed reduction (p = 0.026); screw and plate fixation was preferable to cannulated screw or Kirschner wire (K-wire) fixation for decreasing the rate of AVN in patients treated by open reduction (p = 0.034). Conclusion The rate of AVN increases with age, especially in patients aged 12 years or greater, and with the severity of displacement. In patients treated by closed reduction, anatomical reduction helps to decrease the rate of AVN, while in those treated by open reduction, screw and plate fixation was preferable to fixation using cannulated screws or K-wires. Cite this article: Bone Joint J 2019;101-B:1160–1167

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LBP promoted the peripheral blood recovery of irradiation or chemotherapy-induced myelosuppressive mice, and the effects may be the result of the stimulation of PBMCs to produce G-CSF.

The equilibrium states of homogeneous turbulent buoyant flows are investigated through a fixed-point analysis of the evolution equations for the Reynolds stress anisotropy tensor and the scaled heat flux vector. The mean velocity and thermal fields are assumed to be two-dimensional. Scalar invariants formed from the Reynolds stress anisotropy tensor, the scaled heat flux vector, and the strain rate and rotation rate tensors are governed by a closed set of algebraic equations derived for the stress anisotropy and scaled heat flux under a (weak) equilibrium assumption. Six equilibrium state variables are identified for the buoyant case and contrasted with the corresponding two state variables obtained for the non-buoyant homogeneous turbulence case. These results, while dependent on the functional forms of the models for the pressure-strain rate correlation tensor and the pressure-scalar-gradient correlation and viscous dissipation vector, can be used as in the non-buoyant case to either calibrate new closure models or validate the performance of existing models. In addition, since the analysis only involves the turbulent time scales (both velocity and thermal) and their ratio, the results of the analysis are independent of the specific models for the dissipation rates of the turbulent kinetic energy and the temperature variance. The analytical results are compared with model predictions as well as recent direct numerical simulation (DNS) data for buoyant shear flows. Good agreement with DNS data is obtained.