Drawing upon the stress buffering model of social support, this study investigated how perceived social support (PSS), defined as the amount of support individuals think they can mobilize from their network, and received social support (RSS), defined as the level of support individuals have received, moderated the direct and indirect relationships between COVID-19 news exposure (i.e., stressor) and stress via social trust. An online survey from six major cities in China (N = 636) revealed that PSS rather than RSS moderated the direct relationship between COVID-19 news exposure and stress such that this relationship was stronger at a low level of PSS than a high level. Additionally, RSS rather than PSS moderated the relationship between COVID-19 news exposure and social trust such that this relationship was stronger at a low level of RSS than a high level. These findings reveal the differential mechanisms by which PSS and RSS function to buffer against stress.

The timing and extent of cell division are crucial for the correct patterning of multicellular organism. In Arabidopsis, root ground tissue maturation involves the periclinal cell division of the endodermis to generate two cell layers: endodermis and middle cortex. However, the molecular mechanism underlying this pattern formation remains unclear.Here, we report that phytohormone brassinosteroid (BR) and redox signal hydrogen peroxide (H 2 O 2 ) interdependently promote periclinal division during root ground tissue maturation by regulating the activity of SHORT-ROOT (SHR), a master regulator of root growth and development.BR-activated transcription factor BRASSINAZOLE RESISTANT1 (BZR1) directly binds to the promoter of SHR to induce its expression, and physically interacts with SHR to increase the transcripts of RESPIRATORY BURST OXIDASE HOMOLOGs (RBOHs) and elevate the levels of H 2 O 2 , which feedback enhances the interaction between BZR1 and SHR. Additionally, genetic analysis shows that SHR is required for BZR1-promoted periclinal division, and BZR1 enhances the promoting effects of SHR on periclinal division.Together, our finding reveals that the transcriptional module of BZR1-SHR fine-tunes periclinal division during root ground tissue maturation in response to hormone and redox signals.

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