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The restarted GMRES algorithm proposed by Saad and Schultz [SIAM J. Sci. Statist. Comput., 7 (1986), pp. 856-869] is one of the most popular iterative methods for the solution of large linear systems of equations Ax = b with a nonsymmetric and sparse matrix. This algorithm is particularly attractive when a good preconditioner is available. The present paper describes two new methods for determining preconditioners from spectral information gathered by the Arnoldi process during iterations by the restarted GMRES algorithm. These methods seek to determine an invariant subspace of the matrix A associated with eigenvalues close to the origin and to move these eigenvalues so that a higher rate of convergence of the iterative methods is achieved.

Abstract. The irbleigs code is an implementation of an implicitly restarted block-Lanczos method for computing a few selected nearby eigenvalues and associated eigenvectors of a large, possibly sparse, Hermitian matrix A. The code requires only the evaluation of matrix-vector products with A; in particular, factorization of A is not demanded, nor is the solution of linear systems of equations with the matrix A. This, together with a fairly small storage requirement, makes the irbleigs code well suited for large-scale problems. Applications of the irbleigs code to certain generalized eigenvalue problems and to the computation of a few singular values and associated singular vectors are also discussed. Numerous computed examples illustrate the performance of the method and provide comparisons with other available codes.

Models of nutrient-plankton interaction with a toxic substance that inhibits either the growth rate of phytoplankton, zooplankton or both populations are proposed and studied. For simplicity, it is assumed that both nutrient and the toxin have the same constant input and washout rate. The effects of toxin upon the existence, magnitude, and stability of the steady states are examined. Numerical simulations demonstrate that the system can have multiple attractors when phytoplankton's nutrient uptake rate is inhibited by the toxin.

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