A search is reported for excited τ-leptons and leptoquarks in events with two hadronically decaying τ-leptons and two or more jets. The search uses proton-proton (pp) collision data at $$ \sqrt{s} $$ s = 13 TeV recorded by the ATLAS experiment during the Run 2 of the Large Hadron Collider in 2015–2018. The total integrated luminosity is 139 fb−1. The excited τ-lepton is assumed to be produced and to decay via a four-fermion contact interaction into an ordinary τ-lepton and a quark-antiquark pair. The leptoquarks are assumed to be produced in pairs via the strong interaction, and each leptoquark is assumed to couple to a charm or lighter quark and a τ-lepton. No excess over the background prediction is observed. Excited τ-leptons with masses below 2.8 TeV are excluded at 95% CL in scenarios with the contact interaction scale Λ set to 10 TeV. At the extreme limit of model validity where Λ is set equal to the excited τ-lepton mass, excited τ-leptons with masses below 4.6 TeV are excluded. Leptoquarks with masses below 1.3 TeV are excluded at 95% CL if their branching ratio to a charm quark and a τ-lepton equals 1. The analysis does not exploit flavour-tagging in the signal region.
SummaryA case of pulmonary alveolar proteinosis is reported, in the belief that this is the first case to be recognized in New Zealand and possibly in Australasia. This condition, of unknown ætiology, was first described in June, 1958, by Rosen, Castleman and Liebow. This chronic and sometimes fatal illness often begins with a pneumonic episode followed by dyspnœa, cough, chest pain and loss of weight. Characteristically, the symptoms, physical and radiological signs and pathological changes are limited to the lower part of the respiratory tract and the lung parenchyma. Equally characteristically, the physical signs fail to reflect the often gross radiological changes of extensive, pulmonary infiltration. The essential histological feature is the plugging of alveolar and other distal air spaces in the lungs with a proteinaceous material rich in lipids. This patient was ill for approximately four years before making a complete recovery.
The clinical and histological features of 53 cases of adenocarcinoma of the lung are presented. In more than one-third of the patients the tumour was a solitary nodule, raising the possibility that it might be a metastatic focus. Although histological features may suggest an origin from such sources as the kidney, uterus, ovary or bowel, nearly all such cancers are, in fact, primary bronchial tumours. It is seldom justifiable to submit the patient with a nodular adenocarcinoma in the lung to a search for a possible extrathoracic primary neoplasm.Adenocarcinomas comprise about 10% of all primary lung cancers. They tend to occur in the more peripheral parts of the lung and are often small and symptomless when first detected radiologically. The location of these tumours encourages surgical resection. The lower male to female ratio of primary adenocarcinomas of the lung compared with the male preponderanc_ for other forms of lung cancer is notable.The purpose of this paper is to review our experience with this type of cancer, to discuss clinical management, and to indicate the difficulties that may arise when deciding histologically whether the tumours are primary or metastatic.
Signs, by which one may judge when any inward sickness is impending 15 The Distemper of 1 832 20 Inflammation, general principles of 25 Chapter II. Fever-and first of Simple fever 32 Symptomatic fevers 38 Chapter III. Inflammation of the Lungs 42 Catarrh or Cold; Cough, and its effects 47-Epidemic fever, or the distemper 52-Low Fever 55 The Cough-chronic cough 57 Broken wind 61 Chapter IV. Affections of the Stomach-inflammation .... 65 Poisons, effects of, and antidotes 67 Worms-Botts, &c 68 Apoplexy, Staggers, Epilepsy 74 Chapter V. Diseases of the Bowels 78 Inflammation :-scouring, molten grease TJ Colic, gripes, or fret .
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