In this paper, the mechanism of radio frequency (RF) causing fire and explosion directly and indirectly was recommended, studies of RF risks in flammable and explosive environments were summarized, some problems in RF risk researches recently were discussed, study prospects of RF risks in flammable and explosive environments were analyzed. The fact that the mechanisms and conditions of RF igniting combustible gases should be researched by more deeply theories and technological means, and that RF security standards should be laid down and a complete and precise RF security assessment system should be established in flammable and explosive environments was put forward.
Induction of neurogenesis can occur in the hippocampus in response to various pathological conditions, such as Alzheimer's disease. The aim of this study was to investigate the changes that occur in endogenous neural stem cells in response to amyloid beta (Aβ) 25-35-induced neuronal cell damage in organotypic hippocampal slice cultures. Cresyl violet staining and Fluoro-Jade B staining were used to detect neuronal cell damage and changes of mossy fiber terminals were observed by Timm's staining. The immunofl uorescence staining was used to detect the newly generated cells in the subgranular zone (SGZ) of the dentate gyrus with specific marker, 5-bromo-2′deoxyuridine (BrdU), Ki-67, Nestin, and doublecortin (DCX). In compared to control slices, neuronal cell damage was observed and the mossy fi bers were expanded to CA3 area by treatment with Aβ 25-35. Ki-67/Nestin-and BrdU/DCX-positive cells were detected in the SGZ. In conclusion, these results demonstrate that Aβ-induced neuronal damage results in an increase in endogenous neural stem cells in rat hippocampal slice cultures not only for gliosis but also for neurogenesis.
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