The case is reported of a 75 year old woman who presented with recurrent nocturnal episodes of acute pulmonary oedema. The cause was uncertain as she had normal cardiothoracic ratio on chest radiography and normal left ventricular systolic and diastolic function by transthoracic echocardiogram. Another transthoracic echocardiogram was repeated when she was recumbent for an hour and had a full stomach. It showed a striking finding of severe left atrial compression by an external structure. Computed tomography of the thorax showed an intrathoracic mass behind the left atrium causing external compression of the left atrium suggestive of a sliding hiatus hernia. Cardiac catheterisation confirmed the diagnosis by showing a pronounced rise of pulmonary capillary wedge pressure in the recumbent position compared with the sitting up position.A 75 year old woman presented with recurrent episodes of shortness of breath and chest pain in the previous three months requiring multiple admissions. The diagnosis of acute pulmonary oedema was made but no cause could be found on previous admissions. Her cardiothoracic ratio was normal on chest radiography, her left ventricular function, both systolic and diastolic, were normal by transthoracic echocardiogram. Her symptoms occurred typically at bedtime, especially after a heavy dinner, and were associated with orthopnea, paroxysmal nocturnal dyspnea, and ankle oedema. Physical examination showed regular pulses with a normal blood pressure finding of 124/61 mm Hg. The jugular venous pressure was raised, the heard sounds were normal, and no murmur could be heard. There was bilateral ankle oedema as well as basal crackles heard over both lungs. An electrocardiogram showed normal sinus rhythm without any ischaemic or hypertensive changes. Careful examination of the chest radiograph showed congested lung field with mild bilateral pleural effusion compatible with acute pulmonary oedema. There was also a round shadow behind the heart with an air-fluid level within it. Blood tests including complete blood counts, renal and liver function test, and creatinine kinase activity were within normal limits. Transthoracic echocardiography was repeated when the patient was in the supine position for an hour and had a full stomach. It showed normal left ventricular function but the left atrium was severely compressed by an extrinsic structure confirmed by multiple views (fig 1). Spiral computed tomography of the thorax showed a large hiatus hernia with intrathoracic extension. The hernia was located behind the left atrium causing anterior shift of the heart (fig 2). Subsequently coronary angiography showed normal coronary anatomy. Right heart catheterisation showed that baseline right atrial pressure and pulmonary capillary wedge pressure during prolonged supine positioning were 8 mm Hg and 18 mm Hg respectively. However, after sitting upright for 30 minutes, the right atrial pressure and pulmonary capillary wedge pressure decreased to 5 mm Hg and 6 mm Hg respectively, confirming the diagnosis of s...
Hip fractures are common events in the geriatric population and are often associated with significant morbidity and mortality. Over the coming decades, the size of the greying population is forecast to increase and hence, the annual incidence of hip fracture is expected to rise substantially. Several studies have shown that hip fracture surgery performed within 24 to 48 h of hospitalisation significantly reduces mortality. Medical specialists including cardiologists are often involved in the care of these geriatric patients as most of them have comorbid conditions that must be managed concomitantly with their fracture. Cardiovascular and thromboembolic complications are among some of the commonest adverse events that could be experienced by these elderly patients during hospitalisation. We review in this article the current recommendations and controversies on the peri-operative management of anti-platelet agents and anti-thrombotic agents in geriatric patients undergoing semi-urgent hip fracture surgery.
A 50-year-old man with long standing ankylosing spondylitis developed cauda equina syndrome, which was found to be coexistent with a spinal arterio-venous malformation. Paraplegia ensured following an acute exacerbation of back pain along with an attack of uveitis. Vasculitis changes were found on resected abnormal vessels.
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