؉ -depleted animals exhibited decreased antigen-specific lymphocyte proliferative response, an increased antigen-specific production of interleukin-4, and a lack of specific immunoglobulin G2 antibody. This suggests that WC1 ؉ ␥␦ TCR ؉ cells contribute, either directly or indirectly, toward the Th1 bias of the immune response in bovine tuberculosis-a hypothesis supported by the decreased innate production of IFN-␥, which was observed in WC1 ؉ -depleted calves.
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