Letter to the Editor Dear Editor, Epicrania fugax has recently been defined as a paroxysmal headache localized in the posterior cranial area, which starts at a focal area and rapidly radiates ipsilaterally around the eye or the nose (1,2). Autonomic findings can accompany some cases (2,3). In this syndrome, the average onset age of which is 48 years, the pathogenesis has not been fully enlightened. Its treatment is similar to the treatment of neuralgiform pains. In this article, a case who was diagnosed with epicrania fugax, a newly defined primary headache, and who responded to gabapentin (1800 mg/day) treatment was presented. A 59-year-old female patient presented to our neurology polyclinic with a complaint of headache that had persisted for 2 years. The headache was moderate or severe, it lasted 5-10 s, it started from the right side of the back of the head and radiated to the eye, it was a stabbing type of pain, it was accompanied with lachrymation, and its attack frequency was 2-3 times a day. Neurological and other system examinations were normal. There was no risk factor other than hypertension. Contrast brain-diffusion magnetic resonance imaging and magnetic resonance angiography examinations were normal. No pathology was found in the laboratory examinations. The patient did not respond to indomethacin (150 mg/day) and carbamazepine (600 mg/day) treatments. The case whose headache was reconsidered was thought to have epicrania fugax. Our patient who began to have less headaches with the treatment of gabapentin 1800 mg/day is still being followed by our polyclinic. Epicrania fugax was first defined by Pareja et al. (1) in 2008. Although there were only 20 cases defined until 2009, this number has currently increased to 100 (1,2,3). Its characteristics that differentiate it from other epicranias and neuralgiform aches are the origin of the headache, its duration, and how it radiates (4). The type of headache that can be confused the most with epicrania fugax is the short-term, one-sided neuralgiform headache accompanied by autonomic findings; however, this type of headache does not radiate (5). Although its pathogenesis is not fully known, peripheral and central mechanisms are thought to play a role (1,2). Case reports show responses to the treatments of occipital nerve block, carbamazepine, lamotrigine, pregabalin, and gabapentin (1,2,3). In one-sided, short-term headaches that start from a focal region at the back of the head and quickly radiate to the nose and the eye, epicrania fugax, which is a newly defined type of headache, should be considered.
Elin motor temsili presantral girustadır. Bu bölgenin küçük infarktları, izole el felcine neden olabilir. Eldeki güçsüzlük bütün el parmaklarında olabileceği gibi sadece ulnar taraf parmaklarında da olabilir. Yetmiş bir yaşında erkek hasta ani gelişen sağ el 4. ve 5. parmaklarındaki güçsüzlük nedeniyle acil servise başvurmuş. Çekilen difüzyon MR da sağ presantral girus lokalizasyonunda akut enfarkt saptanması üzerine iskemik inme tanısıyla nöroloji servise yatırıldı. İskemik inme subtipi kardiyoembolik inme olarak belirlendi. Bu yazıda ulnar sinir paralizisini taklit eden akut iskemik inme olgusu sunulmuştur. Anahtar Sözcükler: Embolizm, ulnar taraf, presentral girus, küçük kortikal enfarkt. REPORT OF ISCHEMIC STROKE MIMICKING ISOLATED ULNAR NERVE PARALYSIS ABSTRACTThe cortical motor hand area is the precentral gyrus. Small cortical infarcts of this area can caused isolated hand weakness. Weakness can consist of either all fingers or ulnar-sided fingers. A 71-year-old man admitted to the emergency department with sudden weakness of the right fourth and fifth fingers Diffusion-weighted brain imaging of a magnetic resonance imaging scan revealed acute infarction of right precentral gyrus. Cardioembolus is the determined ischemic stroke subtype. This report presented a case of ischemic stroke mimicking isolated ulnar nerve paralysis
Short-lasting unilateral neuralgiform headaches with conjunctival injection and tearing (SUNCT) syndrome is one of the trigeminal autonomic cephalalgias. Headache particularly occurs in the orbital-supraorbital region and lasts for approximately 49 s (1,2). The varicella-zoster virus (VZV) is a member of the herpes virus family, and it can remain latent in the trigeminal nerve ganglion and expand from the ganglion to the sensorial nerve when reactivated (2). The risk of developing herpes zoster virus (HZV) infection in a lifetime is 20% (3). The nerve most affected by HZV is the fifth cranial nerve (particularly, the ophthalmic nerve) (4). This paper presents two cases of herpes zoster ophthalmicus whose headaches in the prodromal period mimicked the SUNCT syndrome and who were diagnosed with the presentation of vesicular desquamation. Informed written consent was obtained from the participants.
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