Background Previous studies show the shape of the femur in developmental dislocation of the hip (DDH) becomes more abnormal with increasing subluxation. Two kinds of high dislocations associated with DDH have been observed in clinical practice, one with (Type C1) and one without (Type C2) a false acetabulum. The presence or absence of a false acetabulum in high dislocated hips is associated with different loading patterns and could influence the development and shape of the proximal femur. Questions/purposes We therefore determined whether (1) the proximal femoral shape and dimension in Type C1 and Type C2 hips differ from each other, and (2) the femur dislocated with the same height in Types C1 and C2 hips. Patients and Methods We examined the following variables on 54 proximal femurs from 54 patients with high DDH (28 Type C1 hips and 26 Type C2 hips) on AP and lateral radiographs; the ML widths of the cortical and medullary canals, height of the femoral head, height of dislocation, and height of the greater trochanter. Reproducibility of the measurements was tested by two researchers with high interobserver and intraobserver agreement.Results The proximal femur in Type C2 hips was narrower and stovepipe shaped, with a smaller flare index (2.7 ± 0.6), compared with Type C1 hips (3.5 ± 1.2). The proximal femur migrated an average of 18 mm more superiorly in Type C2 than in Type C1 hips. Conclusions Our data confirm distinctions in the shape of the proximal femur in the presence and absence of a false acetabulum. Clinical Relevance Owing to the abnormal shapes, special implants of different geometries or modular stems may be needed for reconstruction Type C2 high dislocations.
The quadrant system, although helpful in determining screw placement in hips with a normal center of rotation, can be misleading and of less value in guiding screw insertion to augment acetabular shells for hips with a high dislocation. We believe that a safe zone specific to hips with a high dislocation should be used to guide transacetabular screw fixation.
Myoclonic movement induced by etomidate is a common but undesirable problem during general anesthesia induction. To investigate the influence of pretreatment with low-dose ketamine on the incidence and severity of myoclonus induced by etomidate, 104 patients were randomized allocated to 1 of 2 equally sized groups (n = 52) to receive either intravenous low-dose ketamine 0.5 mg/kg (group K) or an equal volume of normal saline (group S) 1 minute before induction of anesthesia with 0.3-mg/kg etomidate. The incidence and severity of myoclonus were assessed for 2 minutes after administration of etomidate. Here, we found that the incidence and intensity of myoclonus were both significantly reduced in low-dose ketamine-treated group compared with saline-treated group. The incidence of adverse effects was low and similar between groups. These results demonstrate that intravenous infusion of low-dose ketamine 0.5 mg/kg 1 minute prior to etomidate administration is effective in relieving etomidate-induced myoclonic movements during general anesthesia induction.
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