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SummaryThe responses of plasma free fatty acid, glucose, lactate, insulin and growth hormone to intravenous administration of the predominantly P-2 stimulant, salbutamol, were studied in nine normal subjects. Four subjects received the predominantly 1-1 blocking agent, practolol, together with salbutamol.Salbutamol produced a marked rise in free fatty acid levels and there was also an appreciable increase in insulin values; lactate levels rose moderately and glucose values increased slightly; growth hormone levels were not affected. Practolol did not alter the free fatty acid response; it diminished but did not appear to abolish the insulin response, and it appeared to suppress the lactate and glucose rises.Tentative proposals are made regarding the nature of the preceptor subtypes responsible for mediating these effects; most definite is the suggestion that lipolysis is subserved by 1-2 receptors. Possible clinical implications are briefly discussed. Introduction1-adrenergic stimulants can be divided into two types. The first, which is best exemplified by isoprenaline (isoproterenol), are non-selective and,

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Apatitic tricalcium phosphate was used as support for supported aqueous phase catalysis (SAPC) in the hydroformylation reaction of oct‐1‐ene, at 80 °C in toluene using a dinuclear rhodium complex bearing TPPTS as hydrophilic ligands. The reaction yield is maximum and constant when the hydration rate of the support is ranging from 20 to 35%, before starting to decrease dramatically. The support was examined initially and after catalytic runs by XRD, FTIR, 1H and 31P NMR, SEM and EDS. No change was observed when the hydration rate is less than 35%. Beyond 35%, some decomposition of the support into monetite and stoichiometric apatite occurred; the drop in the yield is essentially due to a loss of droplets of water from the support. The outstanding yield observed with the apatitic tricalcium phosphate, when compared to silica usually used in SAPC, was attributed to the orientation of the catalytic molecule arising from the adsorption of some of the TPPTS ligands onto the hydrophilic surface of apatite.

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