Resistin, which is derived from the gene of RSTN, belongs to a family of cysteine-rich secretory proteins called resistin-like molecules (RELMs). Increased serum resistin levels are associated with coronary artery disease (CAD) and the risk of cardiovascular death. Patients (n = 214) with an initial diagnosis of stable angina pectoris, unstable angina pectoris, and myocardial infarction without ST-segment elevation and referred to catheter laboratory for coronary angiography were enrolled in the study. We aimed to investigate the relationship between increased serum resistin level and CAD. The severity of CAD was calculated by the Gensini scoring system. In conclusion, we established a significant correlation between serum resistin levels and CAD (P = .010). Also, serum resistin levels correlated with the Gensini score that represents the severity of CAD angiographically (P = .010).

patterns with or without Q waves on a 12-lead resting electrocardiography (ECG) (1). It indicates heterogeneous depolarization of the ventricular myocardium that can occur owing to ischemia, fibrosis, or scar. Recent studies have shown the relationship of fQRS with various cardiovascular diseases such as coronary heart disease (CHD), dilated cardiomyopathy, heart failure, hypertension, cardiac arrhythmias, and metabolic syndrome (2-9).Although fQRS in lateral leads is shown to be associated with a poor outcome in patients with a known cardiac disease, the knowledge about the significance and prevalence of fQRS especially in inferior leads is scarce (3). A limited number of studies have reported that the prevalence of fQRS in healthy individuals ranges from 7.2% to 19.7%, with the conclusion that fQRS in inferior leads is not associated with poor clinical outcomes, especially in healthy individuals (10, 11).This study aimed to investigate the prevalence and predictors of fQRS in inferior leads in healthy young men.

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SummaryPulmonary embolism (PE) caused by obstruction of the pulmonary arterial bed is an acute, life threatening, cardiovascular emergent situation. It is a reversible cause of right ventricular failure. The initial diagnosis majority may be missed due to symptoms and signs are nonspecific. Because of late detection of diagnosis is fatal, PE should be thought when acute dyspnea occurs. PE and deep vein thrombosis (DVT) are clinical situations related to venous thromboembolism (VTE). PE accompanies 50% to DVT. (1) The real incidence of DVT and PE is not known due to nonspecific clinical situation. Pulmonary embolism occurs in 0.4% of hospitalized patients. (2) Keywords: Pulmonary embolism, deep vein thrombosis, ventricular failure. CaseA 34 years old professional basketball player was admitted to our emergency service with ambulance service due to acute dyspnea. The dyspnea had started suddenly after basketball training and the general condition of the patient has rapidly deteriorated. The patient had an injury of left knee anterior cruciate ligament two month ago. A knee bandage had been advised by an orthopedist. Except this injury there was no special characteristic on his background. On physical examination, he was unconscious and extremely cyanotic. He had severe dyspnea and tachypnea. First and second heart sound were normal, but there was right ventricular third heart sound (S3) and jugular venous distention. The blood pressure was 80/50 mmHg. The ECG