Effects of the crustacean hyperglycemic hormone on total lipids in the hemolymph of Chasmagnathus granulata and on free fatty acids, phospholipids, and triglycerides in the hemolymph of Carcinus maenas, as well as on their release by Orconectes limosus hepatopancreas in vitro, were investigated. Eyestalk ablation led to a significant decrease of total lipids in the hemolymph of C. granulata and of free fatty acid levels in C. maenas. Crustacean hyperglycemic hormone injections completely reversed the effects of eyestalk extirpation in short-term experiments. Crustacean hyperglycemic hormone also caused significant elevations of hemolymph phospholipids and triglycerides in eyestalkless C. maenas. The release of free fatty acids and phospholipids from O. limosus hepatopancreas in vitro was significantly increased in the presence of crustacean hyperglycemic hormone. These results suggest a physiological role for crustacean hyperglycemic hormone in the control of lipid metabolism in crustaceans. This role may be similar to that of the adipokinetic hormone of insects.

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Cellular oxidative stress may promote damage or death in biological systems and may be caused by production of pro-oxidant molecules known as reactive oxygen species (ROS). , 1987), a gill chamber parasite known for its capacity to cause host metabolic changes, including changes in oxygen consumption rates. Infested and non-infested shrimp were collected in the Patos Lagoon estuary (southern Brasil), where the prevalence of the paraslte may be as high as 70%. No significant differences were observed for either CAT or GPx activities However, SOD activity was significantly reduced in lnfected shrimp, suggesting that bopyrid isopod respiratory impairment resulted in reduced SOD enzyme activity