Prior reviews have identified elevated trait anger as a risk factor for intimate partner violence (IPV) perpetration. Given that 10 years have passed since the last comprehensive review of this literature, we provide an updated meta-analytic review examining associations among anger, hostility, internalizing negative emotions, and IPV for male and female perpetrators. One hundred and five effect sizes from 64 independent samples (61 studies) were included for analysis. IPV perpetration was moderately associated with the constructs of anger, hostility, and internalizing negative emotions. This association appeared stronger for those who perpetrated moderate to severe IPV compared to those who perpetrated low to moderate IPV, and did not vary across perpetrator sex, measurement method, relationship type, or perpetrator population. Implications and limitations of findings were reviewed in the context of theoretical models of IPV, and future directions for empirical and clinical endeavors were proposed.
This meta-analysis was the first study of which we are aware to investigate the association between Diagnostic and Statistical Manual of Mental Disorders (4th ed., text rev.; DSM-IV-TR; American Psychiatric Association, 2000) PTSD symptom clusters and parent, child, family, and marital/partner functioning problems (e.g., intimate partner violence [IPV] and intimacy). Of the 23 studies that met inclusion criteria, the sample was predominantly male (83.8%), Caucasian (65.0%), and from the military (98.9%). The average age was 43.65 years old (SD = 6.27); the average sample size was 397.4 (SD = 416.9; total N = 9,935). PTSD symptom clusters were assessed primarily by self-report (87.0%), with 8.7% using a rating by a clinician. We used fixed analysis following Fisher's r to z transformation and an unbiased weighing and summing of effect sizes within samples and across studies. We found a small association between hyperarousal and IPV (z = .20). We also found two moderate associations for the emotional numbing and avoidance symptom clusters: (a) with parent, child, and family functioning (z = .32, z = .28, respectively); and (b) with intimacy problems (z = .35, z = .42, respectively). We found two large associations for emotional numbing: marital and parent problems (z = .47) and parent, child, and family functioning problems (z = .32, respectively). Our findings suggested that treatments aim to lessen the effect on those who have close relationships with the individual with PTSD.
Objective: In the present study, we examined whether intimate partner aggression (IPA) could be predicted by variables corresponding to the I3 model’s “perfect storm” theory (“I-Cubed”; Finkel & Eckhardt, 2013). Accordingly, we investigated whether IPA was predicted by an interaction of three processes: presence of instigatory cues (interpersonal provocation), aggression-impelling traits (dispositional anger), and diminished inhibitory control (poor emotion regulation). Method: Individuals with a history of IPA (N = 180) were randomly assigned to use a specific emotion regulation strategy (cognitive reappraisal, distraction, suppression, or no instruction) while imagining an anger arousing (instigation) or neutral (no instigation) relationship scenario using the Articulated Thoughts in Simulated Situations paradigm. IPA-related behaviors were assessed via participants’ coded aggressive verbalizations during the Articulated Thoughts in Simulated Situation scenarios and self-reported intention to engage in physical aggression. Results: Results supported the “perfect storm” hypothesis that IPA-related outcomes were higher among participants who endorsed high (vs. low) levels of trait anger, experienced provocation, and engaged in suppression as a weak inhibitory strategy for emotion regulation. Conclusions: The use of a comprehensive theoretical framework such as the I3 model for the assessment of interactive risk factors for IPA will aid determining for whom, and under what circumstances, IPA is most likely to occur, and may further inform clinical intervention and social policy.
Background It is well established that individual difference factors modulate aggression under the acute effects of alcohol. In this investigation, we tested the hypothesis that one core dimension of psychopathy, Impulsive Antisociality, would modulate intoxicated aggression, whereas another dimension, Fearless Dominance, would not. Methods Participants were 516 young social drinkers (253 men and 263 women). Psychopathy was measured using the Psychopathic Personality Inventory (PPI; Lilienfeld and Andrews, 1996). Following the consumption of either an alcohol or a placebo beverage, aggression was measured with a task in which participants administered and received electric shocks to/from a fictitious opponent under the guise of a competitive reaction-time task. Results Hierarchical regression analyses supported our hypothesis: Impulsive Antisociality predicted aggression under alcohol, whereas Fearless Dominance did not. Conclusions Persons who tend to endorse antisocial and impulsive externalizing behaviors appear to be at greater risk for aggression under the acute influence of alcohol.
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