Spheroidal carbonaceous particles (SCPs) are produced by the high-temperature combustion of fossil fuels and emitted in to the atmosphere. The distribution and concentration of SCPs in sediments have been used as a proxy for the distribution of pollutants emitted to the atmosphere. However, the effect on the distribution of SCPs due to industrial activity at nearby industrial cities is yet to be quantified. To clarify the origin of SCPs of *20 lm in size that are preserved in sediments, we evaluated the abundance, surface morphology, and chemical composition of SCPs in sediment core samples recovered from industrial cities in Japan (Tokyo, Osaka, and Nagasaki) and China (Beijing), with the cities being located at least *500 km away from each other. Vertical profiles of SCP concentration in sediment cores from the Japanese cities and Beijing are different, reflecting the contrasting industrial histories of the two countries. The SCPs from different cities show contrasting morphological and chemical characteristics, suggesting that *20 lm SCPs in sediments from the industrial cities could represent the local combustion history in detail, as the influx of local SCPs is dominant at such sites.
A 16-year-old male with severe thrombocytopenia and progressive multiple organ infarctions was diagnosed as having catastrophic antiphospholipid syndrome (CAPS) complicated with systemic lupus erythematosus, and was successfully treated with combination of anticoagulants, corticosteroids, plasma exchange, and intravenous cyclophosphamide. Antibodies to phosphatidylserine/prothrombin (PS/PT) complex and cardiolipin (CL)/β(2)-glycoprotein I (β(2)GPI) were simultaneously detected, indicating that the different pathways of both PS/PT and CL/β(2)GPI might be associated with the radical manifestation of CAPS.
A 16-year-old male with severe thrombocytopenia and progressive multiple organ infarctions was diagnosed as having catastrophic antiphospholipid syndrome (CAPS) complicated with systemic lupus erythematosus, and was successfully treated with combination of anticoagulants, corticosteroids, plasma exchange, and intravenous cyclophosphamide. Antibodies to phosphatidylserine/prothrombin (PS/PT) complex and cardiolipin (CL)/β(2)-glycoprotein I (β(2)GPI) were simultaneously detected, indicating that the different pathways of both PS/PT and CL/β(2)GPI might be associated with the radical manifestation of CAPS.
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