ObjectivesTo review treatment and outcomes in patients with congenital cricoid cartilage malformation.MethodsRetrospective analysis of patients with diagnosis of congenital cricoid malformation (CCM) treated in a single tertiary pediatric referral center between 1985 and 2022. Patients were grouped according to the morphology of the cricoid cartilage that was diagnosed during endoscopy. We reviewed the treatment strategy(s), decannulation rate, complications, and functional outcomes.ResultsTwenty‐nine patients were grouped into four morphological subtypes of cricoid cartilage: 10 patients had a hypoplastic cricoid, eight had an elliptic shape, five had severe anterior thickening, and six an accentuated V‐shape posterior cricoid plate. Twenty‐four patients underwent surgery, and five were closely followed up without surgical treatment. Eight patients had a tracheostomy prior to surgery, and the majority had a hypoplastic cricoid. Most patients (20 out of 24) required additional procedures postoperatively to achieve an age‐appropriate airway. Thirteen patients needed endoscopic dilatation(s) and granulation tissue removal; four needed more aggressive treatment, and three patients required revision open surgery. Decannulation was achieved after a median of 4.5 months in all previously tracheostomized patients. Endoscopy at the last follow‐up showed an age‐appropriate airway in 27 patients; 20 patients had normal oral feeding, and 11 patients had a normal voice.ConclusionEndoscopy is important to diagnose CCM and most of the time patients would need only watchful waiting. In this report, surgery was required for patients who continued to remain symptomatic and had a compromised airway. The type of surgery depends on the type of cricoid malformation and the grade of stenosis.Level of Evidence4 Laryngoscope, 2023
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Cet article propose, par une revue de la littérature, une synthèse actualisée de l'évaluation initiale du traitement et du suivi des surdités brusques (SB). La SB est une surdité de perception survenant en moins de 72 heures, et d'au moins 30 dBHL sur 3 fréquences consécutives. De diagnostic clinique, le bilan étiologique vise à dépister les étiologies connues comme un traumatisme acoustique, un accident vasculaire ou un neurinome de l'acoustique. Plusieurs hypothèses physiopathologiques existent pour les SB idiopathiques : virale, vasculaire, pressionnelle et autoimmune. Le traitement est controversé vu la récupération spontanée fréquente, néanmoins la corticothérapie reste largement acceptée. Le pronostic dépend de la sévérité de la perte auditive, l'âge, les vertiges et la forme de la courbe audiométrique. En cas de séquelles auditives, différentes options de réhabilitation auditive sont disponibles. Sudden sensorineural hearing loss : Diagnosis and management in 2021This article proposes, through a literature review, an updated summary of the initial evaluation, treatment and follow-up of sudden sensorineural hearing loss (SSNHL) based on current guidelines. SSNHL occurs within 72h with at least 30dBHL loss over 3 consecutive frequencies. Diagnosis is medical in the emergency setting, the etiological workup aims at treating known causes, while different pathophysiological hypotheses exist for idiopathic SSNHL. Controversy exists regarding optimal management due to frequent spontaneous recovery. However, corticotherapy remains widely accepted. Prognosis depends on initial severity, age, associated vertigo and shape of the audiometric curve. Hearing rehabilitation in proposed for significant residual hearing loss.
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