commonly the right ventricular apex can be observed. 3 At the cellular level, alteration of desmosomes is hypothesized to be associated with myocyte apoptosis and inflammation. [4][5][6] It has been suggested that these changes lead to fibrofatty replacement and result in impaired mechanical and electrical coupling producing conduction delay and associated arrhythmias.ARVC was originally considered as a familial progressive degenerative or dysplastic desmosomal cardiomyopathy with autosomal dominant inheritance and variable penetrance. 7 However, only approximately 50% of patients who
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.