IntroductionPhlegmonous gastritis is a rare complication of endoscopic pro− cedures, or may occur in the fetus [1]. Two cases have been re− ported: one followed India ink marking and the other followed polypectomy [2,3]. Several conditions can predispose to the de− velopment of phlegmonous gastritis, including conditions asso− ciated with reduced immune response, gastrointestinal bleeding, and abdominal surgery [4,5]. We encountered a case of phleg− monous gastritis and report it here because of its rarity. Case ReportA 68−year−old woman was transferred to the Department of In− ternal Medicine because of a gastric adenoma. Her vital signs in the endoscopy room were normal, and all her laboratory test re− sults were normal. Endoscopic examination before endoscopic polypectomy showed an elevated mass (0.7 cm 0.7 cm) at the lesser curvature of the antrum (Figure 1 a). We performed an en− doscopic mucosal resection. Eight hours later, she visited the emergency room because of epigastric pain and fever. She looked acutely ill; her blood pressure was 180/90 mm Hg, pulse 88 beats per minute, and body temperature 38.58 C. Her white blood cell count was 11890/mm 3 and her hemoglobin was 14.9 g/dl. No free air was seen on the chest radiograph.A thickened, edematous gastric wall, especially in the antral area, was found on endoscopy (Figure 1 b), but no ulceration or pus was seen. Abdominopelvic computed tomography revealed dif− fuse thickening of the gastric wall (Figure 1 c). We started anti− biotic treatment about 20 hours after the endoscopic mucosal re− section. On day 4 of her hospital stay, follow−up CT showed im− provement in the gastric−wall thickening and perigastric hazi− ness, and her symptoms were relieved. Enterococcus fecalis was cultured from her gastric secretions. Endoscopic biopsy showed histological evidence of acute and chronic gastritis. Antibiotic treatment was continued until day 14 and she was discharged on day 15.One week later, she developed epigastric pain and watery diar− rhea, accompanied by fever, and she visited the outpatient clinic. She was readmitted and given combined antibiotic treatment. Despite this, her fever persisted. Moreover, she had fluctuating Abstract Phlegmonous gastritis, or acute suppurative gastritis, is a rare, life−threatening condition which is characterized by purulent necrosis and thickening of the gastric wall. A combination of gastrectomy and antibiotic therapy is an effective treatment. We report a rare case of phlegmonous gastritis occurring after endoscopic mucosal resection. The patient was treated initially with antibiotic therapy but eventually required surgery. Case Report 490Downloaded by: Karolinska Institutet. Copyrighted material.

In pediatrics, endoscopic examination was useful for the choice of therapeutic strategy and it would be a standard method for evaluation of gastrointestinal presentation.

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Background and Aim. We aim to elucidate the association of risk factors for atherosclerosis and H. pylori infection according to the promotor polymorphism of the CD14 gene in healthy Korean population. Methods. The patients who visited our hospital for routine health examinations and 266 healthy adults (170 males and 96 females) were enrolled in this study. The promotor polymorphism at −159C/T of the CD14 gene was determined by PCR-restriction fragment length polymorphism analysis. According to genetic polymorphism and H. pylori infection, we analyzed the risk of atherosclerosis. Results. The genotype frequencies were CC 7.9%, CT 45.1%, and TT 47.0%, respectively. There were no differences between specific genotypes of CD14 gene and H. pylori infection rate. As for HDL cholesterol level, there were significant differences among the three genotypes (P < 0.01). In subjects with H. pylori infection, no significant differences were observed between specific genotypes of CD14 gene and the risk factors of atherosclerosis. Conclusion. The promotor polymorphism at −159C/T of the CD14 gene was associated with the risk factor of atherosclerosis in healthy Korean population. However, it was not associated with the rate of H. pylori infection and H. pylori induced atherosclerotic risk.