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has an impact on wound healing, wounds were created in Tlr4 À/À and control mice on a high-fat diet. Tlr4 À/À mice demonstrated improved wound healing at all time points after injury (P < .05; Fig). Conclusions: These studies suggest that T2D, specifically increased levels of the SFA palmitate and its interaction with the TLR4 pathway, induces expression of the epigenetic enzyme JMJD3, and this regulates M4-mediated inflammation.

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