The body of information available on the relationship of postnuptial molt to reproductive functions indicates that, in general, these events are separated in time in birds that have altricial young. This is true of those that live in the tropics as well as the temperate zone, regardless of whether they are migratory or nonmigratory (

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SUMMARY Attenuation of exercise-induced increases in heart rate and cardiac output by chronic f3-adrenergic blockade has been thought to conmpromise benefit of exercise training in patients with coronary artery disease (CAD). To assess this important issue, 35 CAD patients were evaluated by a 3-month walkjogcycle training program: 14 patients received no blocker (group 1), 14 received propranolol,' 30-80 mg/day (group 2), and seven patients'received propFan'olol, 120-240 mg/day (group 3). The extent of CAD, resting heart rate before training blood pressure and V02 max were similar (p = NS) in each group. The maximal exercise heart rate (mean i SD, 147 ± 21 beats/min in group 1 vs 120 ± 10 beats/min in group 2 and 115 12 beats/min in group 3 (both p < 0.05 vs group 1). The V02 max before training was 25 5.0 mI/kg/mm in group 1 vs 23 3.2 ml/kg/min in group 2 and 26 ± 2.8 ml/kg/min in group 3 (allp = NS). Training consisted of three 1-hour periods per week at a heart rate of 70-85% of the maximal pretraining heart rate. In each group, VO2 increased (p < 0.05) after training: group 1, 27%; group 2, 30%; group 3, 46%. The'double product was unchanged after training (p = NS) in each group.These Despite the widespread clinical usage of 3-adrenergic blocking drugs in coronary artery disease and the common practice of prescribing exercise conditioning programs to patients with ischemic heart disease, many of whom are receiving d-blocking agents, objective data concerning the effect of these drugs on the patient's ability to achieve a training effect are lacking. It has been presumed that chronic exercise during therapeutically imposed limitation in heart rate

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