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Background: Raised inflammatory markers are associated with worse outcome after percutaneous coronary interventions (PCI). An increase in the white blood cell (WBC) count is a non-specific response to inflammation. We hypothesised that a raised baseline WBC count would be a predictor of mortality in patients undergoing PCI. Methods: The association between preprocedural WBC count and long term mortality was studied in 7179 patients enrolled in the EPIC, EPILOG, and EPISTENT trials. The end points were the incidence of myocardial infarction at one year, and one and three year mortality. Results: There were 188 deaths and 582 myocardial infarctions at one year. While WBC count was a strong predictor of death at one year, with every increase of 1 k/µl (1×10

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there were seven type II endoleaks (12.7%) and a significant reduction in aneurysm maximum transverse diameter in 70.4%. Five limb occlusions occurred at the 1-year and one at the 2-year follow-up (7.0%). Limb occlusions were more common in group 2 (0% vs 12.2%; P ¼ .03 at 1 year), representing the majority of required reinterventions (97.7% vs 77.2%; P ¼ .017 at 2 years). Comments: This real-world experience using the ANACONDA fenestrated endograft provides previously unavailable information on the midterm performance of the device. A technical success of only 80% when treating extensive disease (to the celiac trunk) is concerning, and suggests that improvements are needed for this cohort of patients. The limb occlusion rate is also of concern, and may again reflect pushing the limits of the device in unfavorable anatomic conditions. Lessons learned by these early investigations will undoubtedly allow improvements and refinements in design and patient selection.

stiffness and pulse wave velocity / Aorta and carotid arteries 137 (0.94 to 1.01) p = 0.096; Obesity OR = 0.47 (0.29 to 1.77) p = 0.003 and Diabetes OR = 2.41 (1.15 -5.05) p = 0.020. Conclusions: According to the results obtained, genetic polymorphisms variables were not in the multivariate analysis equation to determine the increase of the PWV, which can be explained either by being included in the selected variables such as hypertension, or on the other hand, they may not have enough strength to remain in the equation. So, according to this study, PWV has much more to do with behaviors and traditional risk factors than the genetic heritage.P883 Endothelial dysfunction, pulse wave velocity and augmentation index are correlated in subjects with systemic arterial hypertension?