Background:
From the evidence of failed injection-based growth factor therapies, it has been proposed that a
naturally triggered uninterrupted blood circulation of the growth factors would be superior.
Objective:
We seek to stimulate discussions and more research about the possibility of using the already available growth
factors found in the prostate gland and endometrium by starting a novel educable physiology, known as biological
transformations controlled by the mind.
Methods:
We summarized the stretch-gated ion channel mechanism of the cell membrane, and offer several practical
methods that can be applied by anyone, in order to stimulate and enhance the blood circulation of the growth factors from
the seminal fluid to sites throughout the body. This details the practical application of our earlier published studies about
biological transformations.
Results:
A previously reported single-patient case study has been extended, adding more from his personal experiences
continually improving this novel physiological training and extending the ideas from our earlier findings in detail.
Conclusion:
The biological transformation findings demonstrate the need additional research to establish the benefits of
these natural therapies to repair and rejuvenate tissues affected by various chronic diseases or aging processes.
In this review, we propose a holistic approach to understanding cancer as a metabolic disease. Our search for relevant studies in medical databases concludes that cancer cells do not evolve directly from normal healthy cells. We hypothesize that aberrant DNA damage accumulates over time—avoiding the natural DNA controls that otherwise repair or replace the rapidly replicating cells. DNA damage starts to accumulate in non-replicating cells, leading to senescence and aging. DNA damage is linked with genetic and epigenetic factors, but the development of cancer is favored by telomerase activity. Evidence indicates that telomere length is affected by chronic inflammations, alterations of mitochondrial DNA, and various environmental factors. Emotional stress also influences telomere length. Chronic inflammation can cause oxidative DNA damage. Oxidative stress, in turn, can trigger mitochondrial changes, which ultimately alter nuclear gene expression. This vicious cycle has led several scientists to view cancer as a metabolic disease. We have proposed complex personalized treatments that seek to correct multiple changes simultaneously using a psychological approach to reduce chronic stress, immune checkpoint therapy with reduced doses of chemo and radiotherapy, minimal surgical intervention, if any, and mitochondrial metabolic reprogramming protocols supplemented by intermittent fasting and personalized dietary plans without interfering with the other therapies.
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