This study examined variables that were hypothesized to contribute to social justice advocacy among 134 graduate students enrolled in counseling programs, including problem solving skills, worldview, social concern, and political interest. Of these variables, the desire to become involved in social justice advocacy and political interest predicted actual engagement in social justice advocacy. The results also showed that many of the participants presented with low levels of social justice advocacy. Implications for training and research are included.

C. Hetherington (1991) hypothesized that lesbian, gay, and bisexual (LGB) adolescents may experience a “bottleneck effect” in career development because of internal psychological energy focusing on issues surrounding sexual identity. This assertion has not yet been tested, however, in the career development literature. The authors examined the relationship between variables indicative of psychological resources being devoted to managing an LGB identity, social support, and career development. Survey data from 102 LGB youth demonstrated that inner sexual identity conflict and social support predicted unique and shared variance in career maturity and vocational indecision, lending empirical support to the bottleneck hypothesis.

The purpose of the present study was to explore the variability in reliability scores on a commonly used career scale, the Career Decision-Making Self-Efficacy Scale (CDMSE). Reliability generalization was employed to identify typical score reliability, variability of score reliability, and variables explaining this variability. Forty-nine pieces of work were examined, and the results revealed that 41% of them reported score reliability of their own data. Of the five subscales, Problem Solving showed the lowest score reliability. In addition, higher score reliability was associated with age, sample racial/ethnic demographics, and standard deviation of total mean score.

The detection rate for mutations in SLC3A1 and SLC7A9 in children was 54% in the SLC3A1 gene for type I chromosomes and 25% in the SLC7A9 gene for non-type I chromosomes. It was lower than that in 10 further patients with an unclassified cystinuria, although the clinical characterization in the first group was more stringent; additionally, different spectrums of mutations were observed. The lack of detectable mutations in many patients indicates the possibility of other yet unidentified genes involved in cystinuria. We could not correlate the severity of the disease to the type of cystinuria in the pediatric patients.