In cellular wireless communication systems, transmitted power is regulated to provide each user an acceptable connection while limiting the interference seen by other users. Previous work has focused on maximizing the minimum carrier to interference ratio (CIR) or attaining a common CIR over all radio links. However, previous work has assumed the assignment of mobiles to base stations is known and xed. In this work, we integrate power control and base station assignment. In the context of a CDMA system, we consider the minimization of the total transmitted uplink power subject to maintaining an individual target CIR for each mobile. This minimization occurs over the set of power vectors and base station assignments. We show that this problem has special structure and identify synchronous and asynchronous distributed algorithms that nd the optimal power vector and base station assignment.

Machine-to-Machine communication recently becomes a popular issue in 4G Standardization Committee, such as IEEE 802.16p and LTE-A. In an OFDMA cellular network embedded with a M2M system, the energy consumption (EC) minimization problem is formulated with the consideration of transmission and circuit energy consumed. We propose joint massive access control and resource allocation (RA) schemes, which perform machine node grouping, coordinator selection, and coordinator RA, and also determine the proper number of groups under a 2-hop transmission protocol, to minimize total EC in both flat-and frequency-selective fading channel. Numerical results show that proposed schemes can achieve sub-optimal EC.

The rate constants for the NCN + NO reaction have been measured by laser photolysis/laser-induced fluorescence technique in the temperature range of 254-353 K in the presence of He (40-600 Torr) and N2 (30-528 Torr) buffer gases. The NCN radical was produced from the photodissociation of NCN3 at 193 nm and monitored with a dye laser at 329.01 nm. The reaction was found to be strongly positive-pressure dependent with negative-temperature dependence, as was reported previously. The experimental data could be reasonably accounted for by dual-channel Rice-Ramsperger-Kassel-Marcus calculations based on the predicted potential-energy surface using the modified Gaussian-2 method. The reaction is predicted to occur via weak intermediates, cis- and trans-NCNNO, in the 2A" state which crosses with the 2A' state containing more stable cis- and trans-NCNNO isomers. The high barriers for the fragmentation of these isomers and their trapping in the 2A' state by collisional stabilization give rise to the observed positive-pressure dependence and negative-temperature effect. The predicted energy barrier for the fragmentation of the cis-NCNNO (2A') to CN + N2O also allows us to quantitatively account for the rate constant previously measured for the reverse process CN + N2O --> NCN + NO.

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The incidence of obesity and its comorbidities, such as insulin resistance and type II diabetes, are increasing dramatically, perhaps caused by the change in the fatty acid composition of common human diets. Adipose tissue plays a role as the major energy reservoir in the body. An excess of adipose mass accumulation caused by chronic positive energy balance results in obesity. The n-3 polyunsaturated fatty acids (n-3 PUFA), DHA (docosahexaenoic acid) and EPA (eicosapentaenoic acid) exert numerous beneficial effects to maintain physiological homeostasis. In the current review, the physiology of n-3 PUFA effects in the body is delineated from studies conducted in both human and animal experiments. Although mechanistic studies in human are limited, numerous studies conducted in animals and models in vitro provide potential molecular mechanisms of the effects of these fatty acids. Three aspects of n-3 PUFA in adipocyte regulation are discussed: (1) lipid metabolism, including adipocyte differentiation, lipolysis and lipogenesis; (2) energy expenditure, such as mitochondrial and peroxisomal fatty acid β-oxidation; and (3) inflammation, including adipokines and specialized pro-resolving lipid mediators. Additionally, the mechanisms by which n-3 PUFA regulate gene expression are highlighted. The beneficial effects of n-3 PUFA may help to reduce the incidence of obesity and its comorbidities.

Employing pre-trained language models (LM) to extract contextualized word representations has achieved state-of-the-art performance on various NLP tasks. However, applying this technique to noisy transcripts generated by automatic speech recognizer (ASR) is concerned. Therefore, this paper focuses on making contextualized representations more ASRrobust. We propose a novel confusion-aware fine-tuning method to mitigate the impact of ASR errors to pre-trained LMs. Specifically, we fine-tune LMs to produce similar representations for acoustically confusable words that are obtained from word confusion networks (WCNs) produced by ASR. Experiments on the benchmark ATIS dataset show that the proposed method significantly improves the performance of spoken language understanding when performing on ASR transcripts.

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Abstract-Coexistence of multiple radio access technologies (RATs) is a promising paradigm to improve spectral efficiency. This letter presents a game-theoretic network selection scheme in a cognitive heterogeneous networking environment with timevarying channel availability. We formulate the network selection problem as a noncooperative game with secondary users (SUs) as the players, and show that the game is an ordinal potential game (OPG). A decentralized, stochastic learning-based algorithm is proposed where each SU's strategy progressively evolves toward the Nash equilibrium (NE) based on its own actionreward history, without the need to know actions in other SUs. The convergence properties of the proposed algorithm toward an NE point are theoretically and numerically verified. The proposed algorithm demonstrates good throughput and fairness performances in various network scenarios.