Cardiac lipomas are rare and usually benign tumors that often remain asymptomatic throughout one's lifetime. We report a case of a 60-year-old man with a cardiac lipoma diagnosed early in childhood. However, due to the lack of surgical expertise in rural India, the lipoma was not removed. After moving to the United States, he received irregular follow-up with serial chest x-ray and computed tomography, which demonstrated an enlarging lipomatous mass occupying the pericardial space. After remaining asymptomatic for more than 37 years, he presented to the hospital with dyspnea. He underwent a surgical resection but, unfortunately, given the extension of the mass into multiple critical portions of the heart, he ultimately died.
Eustachian valve infective endocarditis is rare and mostly affects intravenous drug abusers and those with implanted medical devices or indwelling central venous catheters. The most commonly identified organism is Staphylococcus aureus. Treatment includes intravenous antibiotics for approximately 6 weeks. We present a case of Staphylococcus aureus Eustachian valve endocarditis in an individual without traditional risk factors.
The pathophysiology of narrow complex dilated cardiomyopathy is not defined, so therapeutic options are limited. By utilising the velocity equation, the pathophysiology of narrow complex cardiomyopathy allows above normal conduction propagation velocities. There are two pathophysiological theories that allow above normal conduction velocities and failure to capture the myocardium: (1)insulating fibres of the conduction system extending beyond the apex and (2) reduction of axon branching. A patient with narrow complex cardiomyopathy was subjected to graded increase in amplitude and pulse width pacing to overcome the failure of native conduction to capture the myocardium. Peak systolic strain maps demonstrated a progressive increase in apical contractility with increasing pulse width and amplitude. Ejection fraction improved from 17% to 31%. Understanding the pathophysiology of narrow complex cardiomyopathy leads to proposed therapies. One potential pacing therapy is multi-lead pacing at high amplitude and pulse width to capture myocardial cells not captured by native conduction.
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