This study suggests that the application of IGF-I to the crush-injured site may expedite the functional recovery of paralyzed muscle by increasing the rate of axon regeneration.
SUMMARYFood intake triggers convulsions in fasted mice and rats treated with antimuscarinic drugs, scopolamine or atropine. Bearing some similarities in triggering factor and manifestations of the seizures in patients with eating-evoked epilepsy, seizures in fasted animals may provide insight into the mechanism(s) of this rare and partially controlled form of reflex epilepsy. KEY WORDS: Antimuscarinic-induced seizures, Eating epilepsy, Fasting, Food deprivation, Food intake.A series of studies have shown that fasted mice and rats treated with antimuscarinics, scopolamine or atropine, develop seizures soon after being allowed to eat ad lib (Enginar et al
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