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In todays' world, more than 220 million people live in a country that is not their own. Many people live transnational lives but the social contract between citizen and state is national. How are people on the move protected and provided for in this new global context? Have institutional sources of social welfare begun to cross borders to meet the needs of transnational individuals? This paper proposes a new Global Social Protection (GSP) research agenda, summarizing what we know and what we need to do moving forward. What protections exist for migrants, how are the organized across borders, who can access them and who gets left out? This working paper defines GSP; introduces the idea of a "resource environment" as a heuristic tool with which to map and analyze variations in GSP over time, through space, and across individuals; and provides empirical examples demonstrating the centrality of GSP for scholars of states, social welfare, development, and migration.

Infection by beet cyst nematodes (BCN, Heterodera schachtii) causes a serious disease of sugar beet, and climatic change is expected to improve the conditions for BCN infection. Yield and yield stability under adverse conditions are among the main breeding objectives. Breeding of BCN tolerant sugar beet cultivars offering high yield in the presence of the pathogen is therefore of high relevance. To identify causal genes providing tolerance against BCN infection, we combined several experimental and bioinformatic approaches. Relevant genomic regions were detected through mapping-by-sequencing using a segregating F2 population. DNA sequencing of contrasting F2 pools and analyses of allele frequencies for variant positions identified a single genomic region which confers nematode tolerance. The genomic interval was confirmed and narrowed down by genotyping with newly developed molecular markers. To pinpoint the causal genes within the nematode tolerance locus, we generated long read-based genome sequence assemblies of the tolerant parental breeding line Strube U2Bv and the susceptible reference line 2320Bv. We analyzed continuous sequences of the locus with regard to functional gene annotation and differential gene expression upon BCN infection. A cluster of genes with similarity to the Arabidopsis thaliana gene encoding nodule inception protein-like protein 7 (NLP7) was identified. Gene expression analyses confirmed transcriptional activity and revealed clear differences between susceptible and tolerant genotypes. Our findings provide new insights into the genomic basis of plant-nematode interactions that can be used to design and accelerate novel management strategies against BCN.

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