The formation and progress of cerebral aneurysm is highly associated with hemodynamic factors and blood flow feature. In this study, comprehensive efforts are done to investigate the blood hemodynamic effects on the creation and growth of the Internal Carotid Artery. The computational fluid dynamic method is used for the visualization of the bloodstream inside the aneurysm. Transitional, non-Newtonian and incompressible conditions are considered for solving the Navier–Stokes equation to achieve the high-risk region on the aneurysm wall. OSI and WSS of the aneurysm wall are compared within different blood flow stages. The effects of blood viscosity and coiling treatment on these factors are presented in this work. Our study shows that in male patients (HCT = 0.45), changing the porosity of coiling from 0.89 with 0.79 would decreases maximum OSI up to 75% (in maximum acceleration). However, this effect is limited to about 45% for female patients (HCT = 0.35).
The capture of neutrally buoyant, sub-Kolmogorov particles at the interface of deformable drops in turbulent flow and the subsequent evolution of particle surface distribution are investigated. Direct numerical simulation of turbulence, phase-field modelling of the drop interface dynamics and Lagrangian particle tracking are used. Particle distribution is obtained considering excluded-volume interactions, i.e. by enforcing particle collisions. Particles are initially dispersed in the carrier flow and are driven in time towards the surface of the drops by jet-like turbulent fluid motions. Once captured by the interfacial forces, particles disperse on the surface. Excluded-volume interactions bring particles into long-term trapping regions where the average surface velocity divergence sampled by the particles is zero. These regions correlate well with portions of the interface characterized by higher-than-mean curvature, indicating that modifications of the surface tension induced by the presence of very small particles will be stronger in the highly convex regions of the interface.
The topology of the incompressible steady three-dimensional flow in a partially filled cylindrical rotating drum, infinitely extended along its axis, is investigated numerically for a ratio of pool depth to radius of 0.2. In the limit of vanishing Froude and capillary numbers, the liquid–gas interface remains flat and the two-dimensional flow becomes unstable to steady three-dimensional convection cells. The Lagrangian transport in the cellular flow is organised by periodic spiralling-in and spiralling-out saddle foci, and by saddle limit cycles. Chaotic advection is caused by a breakup of a degenerate heteroclinic connection between the two saddle foci when the flow becomes three-dimensional. On increasing the Reynolds number, chaotic streamlines invade the cells from the cell boundary and from the interior along the broken heteroclinic connection. This trend is made evident by computing the Kolmogorov–Arnold–Moser tori for five supercritical Reynolds numbers.
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