Eighty-four adults between the ages of 20 and 79 were presented with two types of problem-solving tasks. One was a task that is typically used in problem-solving research and the other was a task composed of practical problems that adults might encounter in their daily lives. Performance on the two types of tasks exhibited different developmental functions across age. Performance on the traditional problem-solving task decreased linearly across the page while performance on the practical problems increased to a peak in the 40- and 50-year-old groups and decreased thereafter. The results indicate that the developmental function obtained for problem-solving during the adult years depends on the type of problems that are presented. While performance on the abstract type of problems typically employed in research may decrease with age during the adult years, performance on practical problems may exhibit a different relationship with age.
Ninety-six individuals between the ages of 20 and 80 were presented with two types of problem-solving tasks. One was a traditional laboratory problem-solving task; the other was composed of a number of practical problems. Three types of practical problems were employed--problems that young adults might encounter in their daily lives, problems that middle-aged adults might encounter, and problems that elderly adults might encounter. On the traditional laboratory task, performance decreased with increasing age. On the practical problems, however, performance increased from the 20- to the 30-year-old group and decreased thereafter with the most drastic decreases occurring in the 60- and 70-year-old groups. When the three types of practical problems were analyzed separately, the performance of the younger adults was better than the performance of older adults on all of the problem-solving tasks, even on the practical problems that were designed specifically to be ones that older adults would be more likely to encounter in their daily lives.
Our studies suggest that hematopoietic regeneration after HCT is associated with increased proliferation and differentiation of primitive progenitors. Increased proliferative stress on stem cells bearing genotoxic damage could contribute to the pathogenesis of t-MDS/AML. Extended follow-up of a larger number of patients is required to confirm whether alterations in progenitor and telomere recovery predict for increased risk of t-MDS/AML.
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