Focal dystonia in young aged patients is considered to be the uncommon clinical presentation, unless a secondary cause is to be considered. Infarcts of the corpus callosum are rare and have not been well documented previously. As for a variety of signs and symptoms due to corpus callosum lesion, focal dystonia can be easily overlooked. The case is approved by ethical committee and explained to the patient with patient approval.
The case study is aimed at providing a more thorough analysis of a case of lateral medullary syndrome presented only with persistent hiccup after eating lunch the study hopes to generate an interest for further studies into the topic and focuses on abnormal unusual presentations of lateral medullary syndrome. Ethical committee has approved this case after taking consent from the patient and explaining to him the importance of the case in clinical practice.
An empty sella occurs due to herniation of the arachnoid through an incompetent diaphragma sellae. Over time, cerebrospinal fluid (CSF) pulsations may enlarge the sella and compress the gland against the floor of the sella. Empty sella syndrome is considered as a less common entity and is usually asymptomatic and an incidental finding. However, it can be a manifestation of increased intracranial pressure and can be occasionally severe. Compression of the pituitary gland may affect function, or traction on the optic chiasm and nerves may cause visual symptoms. An empty sella may be classified as primary when this occurs in persons who have not received pituitary radiation or pituitary surgery, while an empty sella discovered following such procedures is classified as secondary empty sella. In this report, we presented a 41-year-old multiparous patient who presented to us with symptoms of headache and left sided hemi-sensory disturbance. Examination was unrevealing except for the fact that she was obese. On evaluation, she was detected to have impaired blood sugars, dyslipidemia and vitamin insufficiency. MRI brain revealed presence of Empty Sella. Further hormonal analysis was normal. She was treated conservatively and regular follow-up was advised.
Enteric fever is widely prevalent in the tropics. Central nervous system involvement is not rare and reported incidence varies from 5% to 35% [1]. Various well-known neuro-psychiatric manifestations include confusional state, encephalopathy, meningism, convulsions and focal neurological deficits. Acute cerebellar ataxia as an isolated neurological complication of enteric fever is very rare and limited to only a few case reports [2]. Here we report a case of enteric fever who presents Acute Cerebellar Ataxia.
This study was conducted to induce experimental diabetes in donkeys and assess some serum biochemical changes in diabetes mellitus.Fifteen donkeys were used in this experiment and divided into two groups:A. Treatment group which include (11) donkeys . Diabetic group. denoted by Dm. B. Control group which includes (4) donkeys denoted by Cm. Animals were housed in field .The animals were left tow weeks for adaptation. Diabetes mellitus was induced by i.v injection of alloxan monohydrate at dose rate 100 mg/kg dissolved in 25 ml of normal saline, while the control groups were injected with 25 ml of normal saline. Blood was collected before injection of alloxan monohydrate and after that (10,20.30,40)days to check fasting and random serum glucose.The laboratory tests including serum glucose, serum urea, serum creatinine, serum cholesterol, serum lactate dehydrogenase , We found out the following result according to the periods of the experiments: Serum glucose level was elevated starting from the 1st 10 days period after induction of diabetes mellitus. It reached its peak levels on day (20,30,40). Serum urea, serum creatinine, serum cholesterol, serum lactate dehydrogenase were elevated in diabetic donkeys,in control groups, but there were no significant difference.
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