No abstract

Patients discharged with mild or moderate cerebral dysfunction sustained their risk of neurological worsening within 1 year of cardiac arrest. Old age, Hispanic ethnicity, and discharge disposition of home with out-patient services may be associated with a poor 1 year neurological recovery pattern after hospital discharge from cardiac arrest.

Objective: To compare 1-year all-cause mortality (ACM) and major adverse cardiovascular events (MACE) in cardiac arrest (CA) survivors with and without posttraumatic stress disorder (PTSD) symptomatology at hospital discharge. Design: Prospective, observational cohortSetting: Intensive care units at a tertiary-care center Patients: Adults with return of spontaneous circulation after in-hospital or out-of-hospital CA between 9/2015-9/2017. A consecutive sample of survivors with sufficient mental status to selfreport CA and the subsequent hospitalization induced PTSD symptoms (CA-induced PTSS) at hospital discharge were included. Interventions: NoneMeasurements and Main Results: The combined primary end point was ACM or MACEhospitalization for nonfatal myocardial infarction (MI), unstable angina (UA), congestive heart failure (CHF), emergency coronary revascularization, or urgent implantable defibrillators/ permanent pacemaker (ICD/PPM) placements within 12 months of discharge. An in-person PTSD symptomatology was assessed at hospital discharge via the PTSD Checklist -Specific (PCL-S) scale; a suggested diagnostic cut-off of 36 for specialized medical settings was adopted. Outcomes for patients meeting (vs. not meeting) this cutoff were compared using Cox-hazard regression

Cortical and subcortical myoclonus are seen in every sixth patient with cardiac arrest and cannot be distinguished using clinical criteria. Either condition may have good functional outcomes.