Noise barriers are the most widespread solution to mitigate noise produced by the continuous growth of vehicular traffic, thus reducing the large number of people exposed to it and avoiding unpleasant effects on health. However, conventional noise barriers present the well-known issues related to the diffraction at the edges which reduces the net insertion loss, to the reflection of sound energy in the opposite direction, and to the complaints of citizens due to the reduction of field of view, natural light, and air flow. In order to avoid these shortcomings and maximize noise abatement, recent research has moved toward the development of sonic crystals as noise barriers. A previous review found in the literature was focused on the theoretical aspects of the propagation of sound through crystals. The present work on the other hand reviews the latest studies concerning the practical application of sonic crystal as noise barriers, especially for road traffic noise mitigation. The paper explores and compares the latest developments reported in the scientific literature, focused on integrating Bragg’s law properties with other mitigation effects such as hollow scatterers, wooden or recycled materials, or porous coating. These solutions could increase the insertion loss and frequency band gap, while inserting the noise mitigation action in a green and circular economy. The pros and cons of sonic crystal barriers will also be discussed, with the aim of finding the best solution that is actually viable, as well as stimulating future research on the aspects requiring improvement.

A common source of variance (i.e., "general intelligence") underlies an individual's performance across diverse tests of cognitive ability, and evidence indicates that the processing efficacy of working memory may serve as one such source of common variance. One component of working memory, selective attention, has been reported to co-vary with general intelligence, and dopamine D1 signaling in prefrontal cortex can modulate attentional abilities. Based on their aggregate performance across five diverse tests of learning, here we characterized the general cognitive ability (GCA) of CD-1 outbred mice. In response to a D1 agonist (SKF82958, 1 mg/kg), we then assessed the relationship between GCA and activation of D1 receptor (D1R)-containing neurons in the prelimbic region of the medial prefrontal cortex, the agranular insular cortex, and the dorsomedial striatum. Increased activation of D1R-containing neurons in the prelimbic cortex (but not the agranular insular cortex or dorsomedial striatum) was observed in animals of high GCA relative to those of low GCA (quantified by c-Fos activation in response to the D1 agonist). However, a Western blot analysis revealed no differences in the density of D1Rs in the prelimbic cortex between animals of high and low GCA. Last, it was observed that working memory training promoted an increase in animals' GCA and enhanced D1R-mediated neuronal activation in the prelimbic cortex. These results suggest that the sensitivity (but not density) of D1Rs in the prelimbic cortex may both regulate GCA and be a target for working memory training.

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In both humans and mice, performance on tests of intelligence or general cognitive ability (GCA) is related to dopamine D1 receptor-mediated activity in the prelimbic cortex, and levels of DRD1 mRNA predict the GCA of mice. Here we assessed the turnover rate of D1 receptors as well as the expression level of the D1 chaperone protein (DRiP78) in the medial PPC (mPFC) of mice to determine whether rate of receptor turnover was associated with variations in the GCA of genetically heterogeneous mice. Following assessment of GCA (aggregate performance on four diverse learning tests) mice were administered an irreversible dopamine receptor antagonist (EEDQ), after which the density of new D1 receptors were quantified. GCA was positively correlated with both the rate of D1 receptor recovery and levels of DRiP78. Additionally, the density of D1 receptors was observed to increase within 60 min (or less) in response to intense demands on working memory, suggesting that a pool of immature receptors was available to accommodate high cognitive loads. These results provide evidence that innate general cognitive abilities are related to D1 receptor turnover rates in the prefrontal cortex, and that an intracellular pool of immature D1 receptors are available to accommodate cognitive demands.