The Bay of Bengal is traditionally considered to be a less productive basin compared to the Arabian Sea. Despite the contrasting chlorophyll and primary productivity pattern, sediment trap data shows that annual fluxes of organic carbon reach comparable rates in both the basins. The traditional mechanisms of nutrient supply to the upper ocean waters cannot account for this. We propose eddy pumping as a possible mechanism of vertical transfer of nutrients across the halocline to the oligotrophic euphotic zone during summer monsoon when upper ocean is highly stratified. This would induce rapid biological uptake and in turn significantly increase biological production. In the northern Bay, riverine input acts as an additional source of nutrients and augments the subsurface nutrient injection to the euphotic zone by eddy pumping. Notwithstanding this, the lower than expected primary production in the north suggests the possible role of riverine sediment in limiting the sunlight for photosynthesis.
Situated in similar latitudes and subjected to similar atmospheric forcing, the tropical basins of the Arabian Sea looses fresh water due to excess evaporation over precipitation while Bay of Bengal receives freshwater via excess rain and river run off. The hydrological imbalance thus created on an annual scale will have to be balanced by the inter‐basin exchange. In winter this happens through the intrusion of Bay of Bengal waters into the Arabian Sea, when the southward flowing East India Coastal Current carrying low salinity waters from the northern Bay feeds into the West India Coastal Current flowing north along the shelf in the Arabian Sea. Advection of nutrients by this intrusion triggers enhanced levels of chlorophyll near the southern part of the western shelf of India and may play a role in altering the biogeochemistry of this intense hypoxic region.
When compared with the general population, the diabetic population is at higher risk of cardiovascular disease (CVD), as predicted by the Framingham Risk Score calculations (10-year risk 20%). For this reason diabetes is considered a “coronary disease equivalent” condition, as classified by the National Cholesterol Education Program Adult Treatment Panel (NCEP-ATP) III. Furthermore, patients with diabetes who experience a myocardial infarction have a poorer prognosis than nondiabetic patients, which contributes to their overall higher mortality. Dyslipidemia is a major underlying risk factor contributing to the excess CVD risk, and is usually more atherogenic in the presence of diabetes. It is uniquely manifested by raised levels of triglycerides, low levels of high-density lipoprotein cholesterol, and smaller, denser, and more atherogenic low-density lipoprotein particles. Recent trials have suggested the need for more aggressive treatment of dyslipidemia in this subpopulation than the current recommendations by the NCEP-ATP III. This review addresses the newer developments in the diabetes arena in terms of our current understanding of atherogenic dyslipidemia in diabetes and data from the latest randomized trials addressing its management.
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