Many large earthquakes are preceded by one or more foreshocks, but it is unclear how these foreshocks relate to the nucleation process of the mainshock. On the basis of an earthquake catalog created using a waveform correlation technique, we identified two distinct sequences of foreshocks migrating at rates of 2 to 10 kilometers per day along the trench axis toward the epicenter of the 2011 moment magnitude (M(w)) 9.0 Tohoku-Oki earthquake in Japan. The time history of quasi-static slip along the plate interface, based on small repeating earthquakes that were part of the migrating seismicity, suggests that two sequences involved slow-slip transients propagating toward the initial rupture point. The second sequence, which involved large slip rates, may have caused substantial stress loading, prompting the unstable dynamic rupture of the mainshock.

Slow earthquakes are characterized by a wide spectrum of fault slip behaviors and seismic radiation patterns that differ from those of traditional earthquakes. However, slow earthquakes and huge megathrust earthquakes can have common slip mechanisms and are located in neighboring regions of the seismogenic zone. The frequent occurrence of slow earthquakes may help to reveal the physics underlying megathrust events as useful analogs. Slow earthquakes may function as stress meters because of their high sensitivity to stress changes in the seismogenic zone. Episodic stress transfer to megathrust source faults leads to an increased probability of triggering huge earthquakes if the adjacent locked region is critically loaded. Careful and precise monitoring of slow earthquakes may provide new information on the likelihood of impending huge earthquakes.

Silent-slip events have been detected at several subduction zones, but the cause of these events is unknown. Using seismic imaging, we detected a cause of the Tokai silent slip, which occurred at a presumed fault zone of a great earthquake. The seismic image that we obtained shows a zone of high pore fluid pressure in the subducted oceanic crust located down-dip of a subducted ridge. We propose that these structures effectively extend a region of conditionally stable slips and consequently generate the silent slip.

[1] We show fine-scale variations of seismic velocities and converted teleseismic waves that reveal the presence of zones of high-pressure fluids released by progressive metamorphic dehydration reactions in the subducting Philippine Sea plate in Tokai district, Japan. These zones have a strong correlation with the distribution of slow earthquakes, including long-term slow slip (LTSS) and low-frequency earthquakes (LFEs). Overpressured fluids in the LTSS region appear to be trapped within the oceanic crust by an impermeable cap rock in the fore-arc, and impede intraslab earthquakes therein. In contrast, fluid pressures are reduced in the LFE zone, which is deeper than the centroid of the LTSS, because there fluids are able to infiltrate into the narrow corner of the mantle wedge, leading to mantle serpentinization. The combination of fluids released from the subducting oceanic crust with heterogeneous fluid transport properties in the hanging wall generates variations of fluid pressures along the downgoing plate boundary, which in turn control the occurrence of slow earthquakes. Citation: Kato, A., et al. (2010), Variations of fluid pressure within the subducting oceanic crust and slow earthquakes, Geophys.

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The precentral gyrus (M1) is a representative target for electrical stimulation therapy of pain. To date, few researchers have investigated whether pain relief is possible by stimulation of cortical areas other than M1. According to recent reports, repetitive transcranial magnetic stimulation (rTMS) can provide an effect similar to that of electrical stimulation. With this in mind, we therefore examined several cortical areas as stimulation targets using a navigation-guided rTMS and compared the effects of the different targets on pain. Twenty patients with intractable deafferentation pain received rTMS of M1, the postcentral gyrus (S1), premotor area (preM), and supplementary motor area (SMA). Each target was stimulated with ten trains of 10-s 5-Hz TMS pulses, with 50-s intervals in between trains. Intensities were adjusted to 90% of resting motor thresholds. Thus, a total of 500 stimuli were applied. Sham stimulations were undertaken at random. The effect of rTMS on pain was rated by patients using a visual analogue scale (VAS) and the short form of the McGill Pain Questionnaire (SF-MPQ). Ten of the 20 patients (50%) indicated that stimulation of M1, but not other areas, provided significant and beneficial pain relief (p<0.01). Results indicated a statistically significant effect lasting for 3 hours after the stimulation of M1 (p<0.05). Stimulation of other targets was not effective. The M1 was the sole target for treating intractable pain with rTMS, in spite of the fact that M1, S1, preM, and SMA are located adjacently.

The authors found that incidentally discovered meningiomas did not always follow an exponential growth pattern but often exhibited more complex patterns of growth. Serial monitoring of tumor volumes and regression analysis may reveal the growth pattern of incidental meningiomas and provide information useful for determining treatment strategy.