[Purpose] The present study aimed to find out the scoliosis prevalence 11–15 years old
children and to create awareness about scoliosis. [Subjects and Methods] All of the
children were assessed using the Adams Forward Bendings Test and a scoliometer. Sagittal
plane changes such as kyphosis, lordosis, hypokyphosis, hypolordosis and anterior head
tilt were screened. Children with trunk rotation angles (ATR) of 4 degrees or more were
suspected of having scoliosis, and were evaluated for a second time for gibbosity height,
arm-trunk distance, and ATR. [Results] A total of 2,207 children were screened and the
evaluation revealed there were 11 girls (0.49%) with a Cobb angle of 10 degrees and more.
The maximum Cobb angle was 43° (right thoracic-left lumbar) and the maximum ATR was 12°.
Two children had kyphosis and lordosis, and one had hypokyphosis and was diagnosed as
having idiopathic scoliosis. [Conclusion] Families should regularly check their children,
even if they are not diagnosed as having scoliosis in school screenings. It is our opinion
that our study increased the awareness of the families about scoliosis by screening,
brochures and posters. In the future, if school screenings were performed as a routine
procedure and scoliotic students were followed over the long term, the actual
effectiveness of screening would be able to be detected.
ObjectiveThe goal of the present study was to investigate the relationship between iliopsoas muscle group weakness and related hip joint velocity reduction and stiff-knee gait (SKG) during walking in healthy individuals.MethodsA load of 5% of each individual's body weight was placed on non-dominant thigh of 15 neurologically intact, able-bodied participants (average age: 22.4 ± 0.81 years). For 33 min (135 s × 13 repetitions × 5 s rest), a passive stretch (PS) was applied with the load in place until hip flexor muscle strength dropped from 5/5 to 3+/5 according to manual muscle test. All participants underwent gait analysis before and after PS to compare sagittal plane hip, knee, and ankle kinematics and kinetics and temporo–spatial parameters. Paired t-test was used to compare pre- and post-stretch findings and Pearson correlation coefficient (r) was calculated to determine strength of correlation between SKG parameters and gait parameters of interest (p < 0.05).ResultsReduced hip flexion velocity (mean: 21.5%; p = 0.005) was a contributor to SKG, decreasing peak knee flexion (PKF) (−20%; p = 0.0008), total knee range (−18.9%; p = 0.003), and range of knee flexion between toe-off and PKF (−26.7%; p = 0.001), and shortening duration between toe-off to PKF (−16.3%; p = 0.0005).ConclusionThese findings verify that any treatment protocol that slows hip flexion during gait by weakening iliopsoas muscle may have great potential to produce SKG pattern combined with reduced gait velocity.
Background: Increased femoral anteversion (IFA) causes functional problems (i.e., tripping, frequently falling, and fatigue) by affecting the pelvis and lower extremity biomechanics. In the frontal plane, increased contralateral pelvic drop and ipsilateral hip adduction, which are mainly considered deteriorated hip abductor muscle mechanisms, are associated with hip and knee injuries. Aims: The aim of this study was to examine the effects of femoral anteversion on hip abductor weakness and frontal plane pelvis–hip biomechanics during walking. Methods: The study included nine subjects with increased femoral anteversion and a control group of eleven subjects. Maximum isometric voluntary contraction (MIVC) values of the hip abductor muscles were measured with a handheld dynamometer. Three-dimensional gait analysis was performed for kinetic, kinematic, and temporo-spatial gait parameters. Non-parametric tests were used for statistical analysis (p < 0.05). Results: There was no significant difference found between the MIVC values of the IFA and control groups (p = 0.14). Moreover, no significant difference was determined between the ipsilateral peak hip adduction (p = 0.088) and contralateral pelvic drop (p = 0.149) in the stance phase. Additionally, there was no correlation between the peak hip adduction angle in the stance phase and normalized MIVC values in the IFA group (r = −0.198, p = 0.44), or in the control group (r = −0.174, p = 0.55). The deviations of pelvic rotation (p = 0.022), hip internal rotation (p = 0.003), and internal foot progression (p = 0.022), were found to be higher in the IFA group than in the controls. Conclusions: IFA may not be associated with hip abductor muscle weakness, and it may not lead to the hip adduction and pelvic depression that can be seen in hip abductor weakness. Increased pelvic rotation and internal hip rotation during walking might be considered as a compensation for the femoral head–acetabulum alignment mechanism in the frontal plane.
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