The ameloblastoma (AB) is a true neoplasm of enamel organ type tissue which does not undergo diff erentiation to the point of enamel formation. AB is locally invasive and recurs despite adequate surgical removal. It is of varied origin, although the stimulus initiating the process is unknown. The study of molecular and genetic alterations associated with the development and progression of the AB will help to predict the course of the tumor and lead to the development of new therapeutic concepts for their management. An attempt has been made at compiling about molecular pathogenesis of AB.
Purpose: The aim of the study is to examine the role of genes and proteins in neurons functioning. Methodology: This study adopted a desktop methodology. This study used secondary data from which include review of existing literature from already published studies and reports that was easily accessed through online journals and libraries. Findings: The study revealed that genes and proteins play pivotal roles in the functioning of neurons, contributing to the complex processes involved in neural development, communication, and plasticity. Genes provide the instructions for the production of proteins, which act as the building blocks and functional molecules within cells. The interplay between genes and proteins is essential for various neuronal processes, including synaptic transmission, neural circuit formation, and the modulation of neuronal activity. Unique Contribution to Theory, Practice and Policy: The study was anchored on the theory of synaptic plasticity which was proposed by Donald Hebb and the theory of neurotransmission which was pioneered by Otto Loewi. The study recommends that policies should be developed to promote genetic screening and testing for neurological disorders, allowing for early interventions and improved outcomes.
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