Zur Lokalisation der Wernicke-Encephalopathie im Kindes- und Erwachsenenalter

Gullotta, F

doi:10.1007/bf00341674

Cited by 6 publications

(1 citation statement)

References 46 publications

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“…Necrotic foci of several millimeters in diameter with prolif erated capillaries, fat granule cells, preserved axons and neurons. Some authors (Sipe, 1973;Jellinger and Seitelberger, 1970;Gullotta, 1968;Guazzi et al, 1968) also point out a proliferation of astrocytes and glial fibrils, which may go beyond the region of the actual lesions. Myelin sheaths within their area are destroyed.…”

Section: Discussionmentioning

confidence: 99%

Subacute Necrotizing Encephalopathy (Leigh) in an Adult

Ulrich¹,

Fankhauser-Mauri²

1978

Eur Neurol

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The case of a 31-year-old man who died after a 4 month illness of adult subacute necrotizing encephalomyelopathy (Leigh) is reported. The disease presented with visual disturbances and the principal symptoms were ptosis, a conjugate ophthalmoparesis and a slight tremor of the hands. The case was misdiagnosed as probable multiple sclerosis. Neuropathology disclosed characteristic symmetrical necrotizing lesions, mainly localized in the brain stem. The similarity of the lesions with Wernicke’s disease is pointed out. Possible etiological and pathogenetic factors are discussed.

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Section: Discussionmentioning

confidence: 99%

Subacute Necrotizing Encephalopathy (Leigh) in an Adult

Ulrich¹,

Fankhauser-Mauri²

1978

Eur Neurol

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Subacute Necrotizing Encephalomyelopathy (LEIGH)

Jellinger

Seitelberger

1970

Ergebnisse Der Inneren Medizin Und Kinderheilkunde

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Juvenile type of subacute necrotizing encephalomyelinopathy (Leigh) with unusual CNS-localisation

Lahl

1981

Acta Neuropathol

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The case of an adolescent girl aged 16 with subacute necrotizing encephalomyelopathy (Leigh) is reported. The chronic course of illness lasting for 7.5 years was characterized by visual and gait disorders progressing to blindness, incomplete spastic tetraparesis, and fatal respiratory insufficiency. Neuropathology, in addition to CNS lesions with typical pattern, revealed involvement of Ammon's horn, fornix, corpora mammillaria, tractus mammillothalamicus, and corpus callosum. The massive damage to the total Ammon's horn formation, the distribution of which correlates to none of the established patterns of lesion, is related to the primary disease given, and an additional secondary transneuronal degeneration of associated systems is suggested.

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Zur Lokalisation der Wernicke-Encephalopathie im Kindes- und Erwachsenenalter

Cited by 6 publications

References 46 publications

Subacute Necrotizing Encephalopathy (Leigh) in an Adult

Subacute Necrotizing Encephalopathy (Leigh) in an Adult

Subacute Necrotizing Encephalomyelopathy (LEIGH)

Juvenile type of subacute necrotizing encephalomyelinopathy (Leigh) with unusual CNS-localisation

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