Zur Frage über den Blutbefund bei epidemischer Encephalitis

Model, M.; Wolf, Anna-Maria

doi:10.1007/bf01652588

Cited by 3 publications

(1 citation statement)

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“…Where significant hematological changes were reported, they were generally in the direction of leucocytosis in acute EL versus leucopenia in influenza (Vaidya 1918; Géronne 1920; Kraus and Pardee 1921; Berger and Untersteiner 1924; Model and Wolf 1925; Dobreff and Saprjanoff 1937). CSF findings offered nothing which alone could secure an EL diagnosis – increased sugar levels, weak pleocytosis, slight protein increase, a colloid pattern reminiscent of weak to moderate lues (but with a negative Wassermann reaction) – but they did aid differential diagnosis with respect to influenza, where CSF findings were generally negative (Grosz and Pappenheim 1919; Boveri 1920; Barré and Reys 1921; Eskuchen 1922; see also Rietti 1935, pp 136–138; Achard 1921, pp 70–83;Stern 1928, pp 231–248).…”

Section: Has Direct Evidence Of Influenza Infection In El Cases Bementioning

confidence: 99%

Encephalitis lethargica and the influenza virus. II. The influenza pandemic of 1918/19 and encephalitis lethargica: epidemiology and symptoms

Foley

2009

J Neural Transm

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This is the first of two papers which critically examine the relationship between the 1918/19 influenza pandemic and encephalitis lethargica (EL). The role of influenza in the etiology of EL was vigorously debated until 1924. It is notable, however, that the unitarian camp were largely reactive in their argumentation; while the influenza skeptics provided detail descriptions of EL and the features they argued to be unique or at least unusual, influenza supporters focused on sequentially refuting the evidence of their opponents. The impression which emerges from this debate is that the individual features identified by the skeptics were not absolutely pathognomic for EL, but, on the other hand, their combination in one disorder had not previously been described for any other disease.

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