2009
DOI: 10.1016/j.bone.2008.08.132
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Worsening of osteonecrosis of the jaw during treatment with sunitinib in a patient with metastatic renal cell carcinoma

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Cited by 100 publications
(56 citation statements)
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“…[55][56][57][58][59][60][61][62] In a limited cohort, Christodolou et al observed 5 cases of ONJ among 116 patients who had been receiving BPs from 2007 to 2008, of which 4 had received bevacizumab or sunitinib, [10] thus suggesting an increased risk of ONJ, which is derived from an association of BPs and innovative agents.…”
Section: Association Of Antingiogenic Drugs and Antiresorptive Agentsmentioning
confidence: 99%
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“…[55][56][57][58][59][60][61][62] In a limited cohort, Christodolou et al observed 5 cases of ONJ among 116 patients who had been receiving BPs from 2007 to 2008, of which 4 had received bevacizumab or sunitinib, [10] thus suggesting an increased risk of ONJ, which is derived from an association of BPs and innovative agents.…”
Section: Association Of Antingiogenic Drugs and Antiresorptive Agentsmentioning
confidence: 99%
“…[89] Besides initial reports of patients also treated with BPs, [10,55,56,58,62,90] sunitinib appeared as the only agent administered in some RCC patients suffering of ONJ [59][60][61] Furthermore, sunitinib was associated to BPs in numerous ONJ cases in RCC population, [67] with an incidence higher than expected when a denominator was reported. [13,57,69] However, Smidt-Hansen reported the possibility of decreasing the ONJ risk after adopting preventive measures (as well as recommended in general BP-treated population).…”
Section: Sunitinibmentioning
confidence: 99%
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“…An analysis of ONJ among patients receiving bevacizumab, docetaxel, thalidomide, and prednisone for metastatic endocrine-resistant prostate cancer suggested an 18% incidence of ONJ [9]. Individual cases of ONJ have been reported in the literature among zoledronate-exposed patients treated with sunitinib for renal cell carcinoma [22,23].…”
Section: Introductionmentioning
confidence: 99%
“…VEGF inhibitors work somewhat more directly to inhibit the osteoclasts through their action on monocytes which then are in the osteoclast developmental feedback loop. Further reading on these medications will be left to the reader for exploration [30][31][32][33][34][35][36][37][38][39][40].…”
Section: History and Medication Review Of Osteochemonecrosismentioning
confidence: 99%