Working memory interacts with emotion regulation to predict symptoms of mania

Reduction in Children’s Impulsivity Enhances Performance in Mathematics, but not English language: Evidence for Impulsive Behaviour Modification Using Cognitive Modelling

“…Research (eg., Franco et al, 2016;Peckham & Johnson, 2019) shows that impulsive behaviours can be reduced using behavioural training strategies. Whereas such strategies appear to have been extensively explored in the Western, Asia and the Oceania, there is a large paucity of research on the use of cognitive behavioural strategies in modifying impulsive behaviour among children in Africa, especially, Ghana.…”

Effects of working memory training on emotion regulation: Transdiagnostic review

“…There have also been some studies in clinical samples. At low levels of working memory ability, low use of suppression has been linked to mania (Peckham, Johnson, & Swerdlow, ). Compared with children without attention deficit hyperactivity disorder (ADHD), children with ADHD displayed increased self‐criticism at high levels of working memory demands, while at low levels of demand they displayed higher levels of positive emotions (Tarle, Alderson, Arrington, & Roberts, ).…”

The longitudinal trajectory of emotion regulation and associated neural activity in patients with bipolar disorder: A prospective fMRI study

“…Poorer executive function further hampers BD patients' daily functioning, 57 which may also have implications for adherence to treatment and has been shown to contribute to poorer clinical course and illness prognosis 58 . Specifically, deficient emotion regulation has been found to interact with poor working memory to predict more mania symptoms at 12‐month follow‐up in patients with BD 25 . Moreover, the DMPFC is associated with mental state inference and is recruited during tasks that require the ability to project oneself outside the present moment and focus on the perspectives and feelings of other people, such as during down‐regulation of emotion using cognitive reappraisal 17,59,60 .…”

“…23 Yet evidence for neuroprogression in BD comes mainly from cross-sectional studies that link greater illness progression to poorer cognitive function. 23,24 Longitudinal studies of emotion regulation have mainly investigated self-reported habitual emotion regulation strategy use as predictors of subsequent mood symptoms at follow-up, for example, Peckham et al, 25 Fletcher et al, 26 Gilbert et al, 27 Alloy et al, 28 Johnson et al, 29 Stange et al, 30 Pavlickova et al 31 These have generally found that maladaptive emotion regulation strategies, such as rumination and self-blame, predicted depressive symptoms at 6-month follow-up 26 and number of depressive episodes at 3.5-year follow-up, 28 whereas the use of adaptive emotion regulation strategies,…”

“… 23 Yet evidence for neuroprogression in BD comes mainly from cross‐sectional studies that link greater illness progression to poorer cognitive function. 23 , 24 Longitudinal studies of emotion regulation have mainly investigated self‐reported habitual emotion regulation strategy use as predictors of subsequent mood symptoms at follow‐up, for example, Peckham et al, 25 Fletcher et al, 26 Gilbert et al, 27 Alloy et al, 28 Johnson et al, 29 Stange et al, 30 Pavlickova et al 31 These have generally found that maladaptive emotion regulation strategies, such as rumination and self‐blame, predicted depressive symptoms at 6‐month follow‐up 26 and number of depressive episodes at 3.5‐year follow‐up, 28 whereas the use of adaptive emotion regulation strategies, such as cognitive reappraisal, predicted declines in depression at 12‐months follow‐up. 29 Nevertheless, long‐term prospective studies of the neural basis of emotion regulation in BD are critically needed to clarify whether or not BD is marked by neuroprogression and elucidate the trajectory of aberrant neural activity during emotion regulation.…”