WHO/ILO work-related burden of disease and injury: Protocol for systematic reviews of exposure to occupational noise and of the effect of exposure to occupational noise on cardiovascular disease

Teixeira, Liliane Reis; Azevedo, Tatiana M.; Bortkiewicz, Alicja; Silva, Denise T. Corrêa da; Abreu, Wagner de; Almeida, Márcia Soalheiro de; Araujo, Marco A.N. de; Gadzicka, Elżbieta; Ivanov, Ivan D.; Leppink, Nancy; Macedo, Marta Ribeiro Valle; Maciel, Elvira M.G. de S.; Pawlaczyk-Łuszczyńska, Małgorzata; Pega, Frank; Prüss‐Üstün, Annette; Siedlecka, Jadwiga; Stevens, Gretchen A; Ujita, Yuka; Braga, José Ueleres

doi:10.1016/j.envint.2018.09.040

Cited by 49 publications

(45 citation statements)

References 48 publications

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“…We also evaluated the entire body of evidence using the Grading of Recommendations, Assessment, Development and Evaluation (GRADE) approach [ 45 ] with Navigation Guide adaptations for observational epidemiological studies [ 46 , 47 ]. Accordingly, we used three levels of evidence certainty (low, moderate, and high), and initially rated the body evidence as having moderate quality if the evidence only came from observational studies and high if there was evidence from randomized studies.…”

Section: Methodsmentioning

confidence: 99%

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Occupational Noise and Hypertension Risk: A Systematic Review and Meta-Analysis

Bolm‐Audorff

Hegewald

Pretzsch

et al. 2020

IJERPH

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A number of epidemiological studies report an association between occupational noise exposure and arterial hypertension. Existing systematic reviews report conflicting results, so we conducted an updated systematic review with meta-analysis. We registered the review protocol with PROSPERO (registration no.: CRD 42019147923) and searched for observational epidemiological studies in literature databases (Medline, Embase, Scopus, Web of Science). Two independent reviewers screened the titles/abstracts and full texts of the studies. Two reviewers also did the quality assessment and data extraction. Studies without adequate information on recruitment, response, or without a comparison group that was exposed to occupational noise under 80 dB(A) were excluded. The literature search yielded 4583 studies, and 58 studies were found through hand searching. Twenty-four studies were included in the review. The meta-analysis found a pooled effect size (ES) for hypertension (systolic/diastolic blood pressure ≥140/90 mmHg) due to noise exposures ≥80 dB(A) of 1.81 (95% CI 1.51–2.18). There is no substantial risk difference between men and women, but data concerning this question are limited. We found a positive dose-response-relationship: ES = 1.21 (95% CI 0.78–1.87) ≤ 80 dB(A), ES = 1.77 (95% CI 1.36–2.29) > 80–≤85 dB(A), and ES = 3.50 (95% CI 1.56–7.86) > 85–≤90 dB(A). We found high quality of evidence that occupational noise exposure increases the risk of hypertension.

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Section: Methodsmentioning

confidence: 99%

“…We summed the downgrading levels first, and then the upgrading levels to determine the final decision. Otherwise, we applied the rules for assessing evidence outlined in Appendix D of Teixeira et al [ 46 ] for our decisions.…”

Section: Methodsmentioning

confidence: 99%

Occupational Noise and Hypertension Risk: A Systematic Review and Meta-Analysis

Bolm‐Audorff

Hegewald

Pretzsch

et al. 2020

IJERPH

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“…Using the cut-off value of 85 dBA, subjects were divided into high-exposure (field workers exposed to ≥ the median (85 dBA)), medium-exposure (field workers exposed to < 85 dBA), and low-exposure (office workers) groups, based on the noise exposure assessment, using a statistical approach. The cut-off value of 85 dBA is an important limit for occupational safety legislation and regulations and is often adopted in other studies 9,11 . The classification into groups was due to the large variations in median noise levels and exposure ranges at the nine frequencies, as shown in Supplemental Figure S1.…”

Section: Methodsmentioning

confidence: 99%

Occupational noise exposure and its association with incident hyperglycaemia: a retrospective cohort study

Chang

Liu

et al. 2020

Sci Rep

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noise pollution is reported to be associated with diabetes, but few studies have elucidated the associations between noise frequency characteristics. We aimed to evaluate the relationships between different noise frequency components and incident hyperglycaemia. An industry-based cohort of 905 volunteers was enrolled and followed up to 2012. Octave-band frequencies of workstation noise and individual noise levels were measured in 2012 to classify subjects' exposures retrospectively. We applied Cox regression models to estimate the relative risk (RR) of hyperglycaemia. An increased RR for hyperglycaemia of 1.80 (95% confidence interval [CI]: 1.04, 3.10) was found among subjects exposed to ≥ 85 A-weighted decibels (dBA) compared with those exposed to < 70 dBA. The highexposure groups at frequencies of 31.5, 63, 125, 250, 500, 1000, and 2000 Hz had a significantly higher risk of hyperglycaemia (all p values < 0.050) than the low-exposure groups. A 5-dB increase in noise frequencies at 31.5, 63, 125, 250, 500 Hz, and 1000 Hz was associated with an elevated risk of hyperglycaemia (all p values < 0.050), with the highest value of 1.27 (95% CI: 1.10, 1.47) at 31.5 Hz (p = 0.001). Occupational noise exposure may be associated with an increased incidence of hyperglycaemia, with the highest risk observed at 31.5 Hz. Growing numbers of field studies have reported an association between occupational noise exposure and cardiovascular disease (CVD) morbidity and mortality 1-10. In 2019, the World Health Organization (WHO) and the International Labour Organization (ILO) provided a joint methodology (WHO/ILO joint methodology) for estimating the work-related burden of CVD and injury due to workplace noise exposure 11. Noise exposure is regarded as an environmental stressor through direct (i.e., sleep disturbance) and indirect (i.e., annoyance) pathways that pose adverse health effects. Acute noise exposure may activate the hypothalamus-pituitary-adrenal axis and the sympathetic-adrenal-medulla axis to elevate the levels of stress hormones, including cortisol, catecholamine, adrenalin, and noradrenalin. Repeated and chronic stimuli may cause overproduction of stress hormones (e.g., cortisol) that increase the levels of fatty acids and glucose to partially restore homeostasis. In addition, catecholamines also boost the energy supply by breaking down triacylglycerol. Such increases in stress hormones may lead to pathophysiologic alterations in blood pressure, blood lipids, blood viscosity, and blood glucose, which promote the development of hypertension, arteriosclerosis, and CVD 12-14. Because the overproduction of cortisol may inhibit pancreatic insulin secretion and reduce insulin sensitivity in the liver, skeletal muscles, and adipose tissue 14 , it is plausible that long-term noise exposure may produce adverse changes in blood glucose. Noise pollution has been reported to be associated with diabetes in many animal and environmental epidemiological studies; however, few studies have elucidated the relationship between noise fre...

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“…Furthermore, several investigations support that inhalation of ambient particulate matter triggers pulmonary and systemic inflammation resulting in metabolic syndrome and cardiopulmonary disease (Clementi et al 2019). Exposure to high occupational noise is also associated with increase in hypertension, cardiovascular disease and cardiovascular mortality (Skogstad et al 2016;Teixeira et al 2019).…”

Section: Smokingmentioning

confidence: 99%

Diet, Lifestyle, Smoking

Tokgözoğlu

Hekimsoy

Costabile

et al. 2020

Handbook of Experimental Pharmacology

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Cardiovascular disease is the leading cause of death globally The past few decades have shown that especially low-and middle-income countries have undergone rapid industrialization, urbanization, economic development and market globalization. Although these developments led to many positive changes in health outcomes and increased life expectancies, they all also caused inappropriate dietary patterns, physical inactivity and obesity. Evidence shows that a large proportion of the cardiovascular disease burden can be explained by behavioural factors such as low physical activity, unhealthy diet and smoking. Controlling these risk factors from early ages is important for maintaining cardiovascular health. Even in patients with genetic susceptibility to cardiovascular disease, risk factor modification is beneficial. Despite the tremendous advances in the medical treatment of cardiovascular risk factors to reduce overall cardiovascular risk, the modern lifestyle which has led to

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WHO/ILO work-related burden of disease and injury: Protocol for systematic reviews of exposure to occupational noise and of the effect of exposure to occupational noise on cardiovascular disease

Cited by 49 publications

References 48 publications

Occupational Noise and Hypertension Risk: A Systematic Review and Meta-Analysis

Occupational Noise and Hypertension Risk: A Systematic Review and Meta-Analysis

Occupational noise exposure and its association with incident hyperglycaemia: a retrospective cohort study

Diet, Lifestyle, Smoking

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